Uteroglobin deficient mice-a novel animal model for IgA nephropathy?
作者信息
Pouria S, Challacombe S J
机构信息
Department of Renal Medicine, Kings College Hospital, UK.
出版信息
Gut. 2000 Apr;46(4):452-3. doi: 10.1136/gut.46.4.452.
Abstract
摘要
相似文献
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本文引用的文献
1
Uteroglobin is essential in preventing immunoglobulin A nephropathy in mice.子宫珠蛋白对预防小鼠免疫球蛋白A肾病至关重要。
Nat Med. 1999 Sep;5(9):1018-25. doi: 10.1038/12458.
2
Uteroglobin: a novel cytokine?子宫珠蛋白：一种新型细胞因子？
Cell Mol Life Sci. 1999 May;55(5):771-87. doi: 10.1007/s000180050331.
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Pathogenesis of immunoglobulin A nephropathy.免疫球蛋白A肾病的发病机制。
Curr Opin Nephrol Hypertens. 1998 May;7(3):247-50. doi: 10.1097/00041552-199805000-00001.
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IgA nephropathy--a disorder of IgA production?
QJM. 1997 Jun;90(6):387-90. doi: 10.1093/qjmed/90.6.387.
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Severe fibronectin-deposit renal glomerular disease in mice lacking uteroglobin.
Science. 1997 May 30;276(5317):1408-12. doi: 10.1126/science.276.5317.1408.
6
IgA nephropathy.
Kidney Int. 1995 Feb;47(2):377-87. doi: 10.1038/ki.1995.50.
7
Glomerulopathy associated with predominant fibronectin deposits: a newly recognized hereditary disease.以纤维连接蛋白沉积为主的肾小球病：一种新认识的遗传性疾病。
Kidney Int. 1995 Jul;48(1):163-70. doi: 10.1038/ki.1995.280.
8
Evidence that the interaction between circulating IgA and fibronectin is a normal process enhanced in primary IgA nephropathy.循环免疫球蛋白A（IgA）与纤连蛋白之间的相互作用是原发性IgA肾病中增强的正常过程的证据。
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