Davidorf F R
Int Ophthalmol Clin. 1975 Fall;15(3):111-24. doi: 10.1097/00004397-197501530-00016.
The surprising finding of viable lymphocytes in clinically inactive scars gives more credence to the immunological theory regarding reactivation of histoplasmosis scars. We feel that the T-lymphocyte theory can explain the mechanism by which an inactive scar becomes active upon reintroduction of the antigen. The clinical course of the disease as well as the response to corticosteroid therapy and photocoagulation are also consistent with this hypothesis.
在临床静止期瘢痕中发现存活淋巴细胞这一惊人发现，为关于组织胞浆菌病瘢痕再激活的免疫学理论提供了更多可信度。我们认为，T淋巴细胞理论可以解释在再次引入抗原后，静止期瘢痕变为活动期的机制。该疾病的临床病程以及对皮质类固醇疗法和光凝治疗的反应也与这一假说相符。
