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The TFPI 536C-->T mutation is not associated with increased risk for venous or arterial thrombosis.

作者信息

González-Conejero R, Lozano M L, Corral J, Martínez C, Vicente V

出版信息

Thromb Haemost. 2000 May;83(5):787-8.

PMID:10823280
Abstract
摘要

相似文献

1
The TFPI 536C-->T mutation is not associated with increased risk for venous or arterial thrombosis.
Thromb Haemost. 2000 May;83(5):787-8.
2
The 536C-->T transition in the human tissue factor pathway inhibitor (TFPI) gene is statistically associated with a higher risk for venous thrombosis.
Thromb Haemost. 1999 Jul;82(1):1-5.
3
The FXIII Val34Leu polymorphism in venous and arterial thromboembolism.静脉和动脉血栓栓塞中凝血因子 XIII Val34Leu 多态性
Haematologica. 2000 Mar;85(3):293-7.
4
Contribution of the cystathionine beta-synthase gene (844ins68) polymorphism to the risk of early-onset venous and arterial occlusive disease and of fasting hyperhomocysteinemia.胱硫醚β-合酶基因（844ins68）多态性对早发性静脉和动脉闭塞性疾病以及空腹高同型半胱氨酸血症风险的影响。
Thromb Haemost. 2000 Oct;84(4):576-82.
5
Prevalence of the C536T mutation in the tissue factor pathway inhibitor (TFPI) gene among patients with venous thromboembolic disease.静脉血栓栓塞性疾病患者组织因子途径抑制物（TFPI）基因C536T突变的患病率。
Thromb Haemost. 2001 May;85(5):938-9.
6
[A new mutation in the prothrombin gene (G20210A) and the risk for venous and arterial thromboembolism].凝血酶原基因新突变（G20210A）与静脉和动脉血栓栓塞风险
Harefuah. 2000 Jul;139(1-2):51-6.
7
The -33T-->C polymorphism in intron 7 of the TFPI gene influences the risk of venous thromboembolism, independently of the factor V Leiden and prothrombin mutations.组织因子途径抑制物（TFPI）基因第7内含子中的-33T→C多态性影响静脉血栓栓塞风险，独立于因子V莱顿突变和凝血酶原突变。
Thromb Haemost. 2002 Aug;88(2):195-9.
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Increased venous versus arterial thrombosis in the Factor V Leiden mouse.在凝血因子V莱顿突变小鼠中静脉血栓形成较动脉血栓形成增加。
Thromb Res. 2007;119(6):747-51. doi: 10.1016/j.thromres.2006.02.014. Epub 2006 Jun 21.
9
Gene polymorphisms implicated in influencing susceptibility to venous and arterial thromboembolism: frequency distribution in a healthy German population.与静脉和动脉血栓栓塞易感性相关的基因多态性：健康德国人群中的频率分布
Thromb Haemost. 2006 Oct;96(4):465-70.
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Factor V Leiden and increased risk for arterial thrombotic disease in young Brazilian patients.莱顿V因子与巴西年轻患者动脉血栓性疾病风险增加
Blood Coagul Fibrinolysis. 2006 Jun;17(4):271-5. doi: 10.1097/01.mbc.0000224846.35001.64.

引用本文的文献

1
Genetic variation of the blood coagulation regulator tissue factor pathway inhibitor and venous thromboembolism among middle-aged and older adults: A population-based cohort study.中老年人群中凝血调节因子组织因子途径抑制物的基因变异与静脉血栓栓塞症：一项基于人群的队列研究。
Res Pract Thromb Haemost. 2022 Nov 9;6(7):e12842. doi: 10.1002/rth2.12842. eCollection 2022 Oct.
2
Study of associated genetic variants in Indian subjects reveals the basis of ethnicity related differences in susceptibility to venous thromboembolism.对印度受试者相关基因变异的研究揭示了种族相关的静脉血栓栓塞易感性差异的基础。
Thrombosis. 2014;2014:182762. doi: 10.1155/2014/182762. Epub 2014 Sep 30.
3
Risk of venous thromboembolism associated with single and combined effects of Factor V Leiden, Prothrombin 20210A and Methylenetethraydrofolate reductase C677T: a meta-analysis involving over 11,000 cases and 21,000 controls.
静脉血栓栓塞风险与因子 V 莱顿、凝血酶原 20210A 和亚甲基四氢叶酸还原酶 C677T 的单一和联合作用相关：一项涉及超过 11000 例病例和 21000 例对照的荟萃分析。
Eur J Epidemiol. 2013 Aug;28(8):621-47. doi: 10.1007/s10654-013-9825-8. Epub 2013 Jul 31.