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本文引用的文献
1
Studies of Porins: Spontaneously Transferred from Whole Cells and Reconstituted from Purified Proteins of Neisseria gonorrhoeae and Neisseria meningitidis.
Biophys J. 1984 Jan;45(1):104-7. doi: 10.1016/S0006-3495(84)84127-2.
2
The role of B/T costimulatory signals in the immunopotentiating activity of neisserial porin.
J Infect Dis. 1999 Sep;180(3):755-61. doi: 10.1086/314966.
3
Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC.
Nature. 1999 Jun 3;399(6735):483-7. doi: 10.1038/20959.
4
Common regulation of apoptosis signaling induced by CD95 and the DNA-damaging stimuli etoposide and gamma-radiation downstream from caspase-8 activation.
J Biol Chem. 1999 May 14;274(20):14255-61. doi: 10.1074/jbc.274.20.14255.
5
Structural and evolutionary inference from molecular variation in Neisseria porins.
Infect Immun. 1999 May;67(5):2406-13. doi: 10.1128/IAI.67.5.2406-2413.1999.
6
The temporal relationship between protein phosphatase, mitochondrial cytochrome c release, and caspase activation in apoptosis.
Exp Cell Res. 1999 Mar 15;247(2):505-13. doi: 10.1006/excr.1998.4380.
7
Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells.
J Biol Chem. 1999 Feb 26;274(9):5654-8. doi: 10.1074/jbc.274.9.5654.
8
Molecular characterization of mitochondrial apoptosis-inducing factor.
Nature. 1999 Feb 4;397(6718):441-6. doi: 10.1038/17135.
9
Distinct caspase cascades are initiated in receptor-mediated and chemical-induced apoptosis.
J Biol Chem. 1999 Feb 19;274(8):5053-60. doi: 10.1074/jbc.274.8.5053.
10
Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.
J Biol Chem. 1999 Jan 22;274(4):2225-33. doi: 10.1074/jbc.274.4.2225.
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