Evans R W
Postgrad Med. 1975 Dec;58(7):79-85. doi: 10.1080/00325481.1975.11714223.
The prognosis in cardiogenic shock remains poor despite improvements in treating other complications of acute myocardial infarction. In some situations, left ventricular function can be improved by increasing the vascular volume, but the benefits of increasing the cardiac uutput must be balanced agains the risk of pulmonary edema. Monitoring of volume therapy is best done via the pulmonary route. The goal of drug therapy is to raise arterial blood pressure and make the heart pump more effectively. An aggressive approach will not cure great numbers of extensive myocardial damage remains, but it will identify those who are hypovolemic or who have other correctable contributory factors. Most important, this approach may help to identify therapies currently in use that may actually increase rather than lessen myocardial damage.
尽管在治疗急性心肌梗死的其他并发症方面有所改善，但心源性休克的预后仍然很差。在某些情况下，增加血容量可改善左心室功能，但增加心输出量的益处必须与肺水肿的风险相权衡。容量治疗的监测最好通过肺部途径进行。药物治疗的目标是提高动脉血压并使心脏更有效地泵血。积极的治疗方法并不能治愈大量存在的广泛性心肌损伤，但它将识别出那些血容量不足或有其他可纠正的促成因素的患者。最重要的是，这种方法可能有助于识别目前正在使用的、实际上可能增加而非减轻心肌损伤的治疗方法。
