Looking for the exit: How do malaria parasites escape from red blood cells?
作者信息
Bannister L H
机构信息
Centre for Neuroscience, King's College London, United Kingdom.
出版信息
Proc Natl Acad Sci U S A. 2001 Jan 16;98(2):383-4. doi: 10.1073/pnas.98.2.383.
Abstract
摘要
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Proc Natl Acad Sci U S A. 2001 Jan 16;98(2):383-4. doi: 10.1073/pnas.98.2.383.
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本文引用的文献
1
Malaria parasite exit from the host erythrocyte: a two-step process requiring extraerythrocytic proteolysis.疟原虫从宿主红细胞中逸出：一个需要红细胞外蛋白水解作用的两步过程。
Proc Natl Acad Sci U S A. 2001 Jan 2;98(1):271-6. doi: 10.1073/pnas.98.1.271.
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A cysteine protease activity from Plasmodium falciparum cleaves human erythrocyte ankyrin.恶性疟原虫的一种半胱氨酸蛋白酶活性可切割人红细胞锚蛋白。
Mol Biochem Parasitol. 2000 Oct;110(2):259-72. doi: 10.1016/s0166-6851(00)00283-8.
3
Release of merozoites from Plasmodium falciparum-infected erythrocytes could be mediated by a non-explosive event.疟原虫感染的红细胞中裂殖子的释放可能由非爆发性事件介导。
Parasitol Res. 1999 Aug;85(8-9):621-4. doi: 10.1007/s004360050606.
4
Cysteine protease inhibitors as chemotherapy for parasitic infections.半胱氨酸蛋白酶抑制剂作为寄生虫感染的化疗药物。
Bioorg Med Chem. 1999 Apr;7(4):639-44. doi: 10.1016/s0968-0896(99)00008-5.
5
Serine protease inhibitors block invasion of host cells by Toxoplasma gondii.丝氨酸蛋白酶抑制剂可阻止刚地弓形虫对宿主细胞的侵袭。
Antimicrob Agents Chemother. 1999 Jun;43(6):1358-61. doi: 10.1128/AAC.43.6.1358.
6
Plasmepsin II, an acidic hemoglobinase from the Plasmodium falciparum food vacuole, is active at neutral pH on the host erythrocyte membrane skeleton.疟原虫蛋白酶II是一种来自恶性疟原虫食物泡的酸性血红蛋白酶，在中性pH条件下对宿主红细胞膜骨架具有活性。
J Biol Chem. 1999 May 14;274(20):14218-23. doi: 10.1074/jbc.274.20.14218.
7
Proteases of malaria parasites: new targets for chemotherapy.疟原虫蛋白酶：化疗的新靶点
Emerg Infect Dis. 1998 Jan-Mar;4(1):49-57. doi: 10.3201/eid0401.980107.
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Human erythroid spectrin alpha subunit and its SH3 domain are sensitive to acidic Plasmodium falciparum proteolytic activity.
C R Acad Sci III. 1996 Nov;319(11):1011-7.
9
A role for erythrocyte band 3 degradation by the parasite gp76 serine protease in the formation of the parasitophorous vacuole during invasion of erythrocytes by Plasmodium falciparum.在恶性疟原虫入侵红细胞过程中，寄生虫gp76丝氨酸蛋白酶介导的红细胞带3降解在寄生泡形成中的作用。
Mol Biochem Parasitol. 1996 Nov 12;82(1):13-24. doi: 10.1016/0166-6851(96)02714-4.
10
The proteases and pathogenicity of parasitic protozoa.寄生原生动物的蛋白酶与致病性
Annu Rev Microbiol. 1993;47:821-53. doi: 10.1146/annurev.mi.47.100193.004133.
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