Gribanov G A
Kosm Biol Aviakosm Med. 1975 Mar-Apr;9(2):9-12.
The effect of "hypoxic" hypothermia on the metabolism of phsopholidpds of the endocrine organia that occurs during acute hypoxia produces no inhibitory effect on the phospholid metabolism in the thyroid, adrenal and testicular glands of the test animals. As a result of distrubed heat release the content of phsopholipids in the thyroid and adrenal glands increases. The rate of the label (P32) incorporation into phospholipids of every gland (especially thyroid and testicular glands) elevates noticeably. This indicates an important compensatory role played by "hypoxic" hypothermia in the phsopholidip metabolism. The mechanimss of these phenomena are discussed.
“低氧性”低温对急性低氧时发生的内分泌器官磷脂代谢的影响，对实验动物甲状腺、肾上腺和睾丸的磷脂代谢没有抑制作用。由于散热障碍，甲状腺和肾上腺中磷脂含量增加。每个腺体（尤其是甲状腺和睾丸）中标记物（P32）掺入磷脂的速率明显升高。这表明“低氧性”低温在磷脂代谢中发挥了重要的代偿作用。本文讨论了这些现象的机制。
