Hagberg G, Hagberg G, Olow I
Acta Paediatr Scand. 1975 Mar;64(2):193-200. doi: 10.1111/j.1651-2227.1975.tb03821.x.
From an unselected series of 560 Swedish cases of cerebral palsy, born 1954-1970, various data of etiologic and pathogenetic interest were analyzed in detail. Untraceable and prenatal factors were found to dominate within the group of spastic hemiplegia. Placental dysfunction in small-for-date babies and severe asphyxia were thought to be the two main pathogenetic factors among the patients with spastic tetraplegia. In spite of a significant decrease in the number of low birth weight children within the group of spastic diplegia, this syndrome was still very characteristic for the child born immature. Ataxic diplegic forms were found to have greater pathogenic similarities to spastic diplegia than to simple ataxia. In two-thirds of the children the latter syndrome was characterized by normal pregnancy, delivery and birth weight and an untraceable (genetic?) factor. Dyskinetic syndromes were mostly encountered after perinatal asphyxia.
对1954年至1970年出生的560例瑞典脑瘫病例进行了非选择性研究，详细分析了各种病因学和发病机制相关的数据。在痉挛性偏瘫组中，无法追溯的因素和产前因素占主导地位。小样儿的胎盘功能不全和严重窒息被认为是痉挛性四肢瘫患者的两个主要发病因素。尽管痉挛性双瘫组中低体重儿数量显著减少，但这种综合征对于未成熟儿出生后仍然非常典型。发现共济失调性双瘫形式与痉挛性双瘫的致病相似性大于单纯共济失调。在三分之二的儿童中，后一种综合征的特征是妊娠、分娩和出生体重正常，且存在无法追溯的（遗传？）因素。运动障碍综合征大多在围产期窒息后出现。
