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The many faces of host responses to tuberculosis.

作者信息

Collins H L, Kaufmann S H

机构信息

Department of Immunology, Max-Planck Institute for Infection Biology, Berlin, Germany.

出版信息

Immunology. 2001 May;103(1):1-9. doi: 10.1046/j.1365-2567.2001.01236.x.

DOI:10.1046/j.1365-2567.2001.01236.x
PMID:11380686
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1783212/
Abstract
摘要

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本文引用的文献

1
MHC class Ia-restricted T cells partially account for beta2-microglobulin-dependent resistance to Mycobacterium tuberculosis.MHC I类分子限制性T细胞部分介导了β2微球蛋白依赖性的对结核分枝杆菌的抗性。
Eur J Immunol. 2001 Jun;31(6):1944-9. doi: 10.1002/1521-4141(200106)31:6<1944::aid-immu1944>3.0.co;2-r.
2
Trafficking and release of mycobacterial lipids from infected macrophages.分枝杆菌脂质从受感染巨噬细胞中的转运与释放
Traffic. 2000 Mar;1(3):235-47. doi: 10.1034/j.1600-0854.2000.010306.x.
3
High frequencies of circulating IFN-gamma-secreting CD8 cytotoxic T cells specific for a novel MHC class I-restricted Mycobacterium tuberculosis epitope in M. tuberculosis-infected subjects without disease.在未发病的结核分枝杆菌感染受试者中，循环中针对一种新型主要组织相容性复合体I类限制性结核分枝杆菌表位分泌干扰素-γ的细胞毒性CD8 T细胞频率较高。
Eur J Immunol. 2000 Sep;30(9):2713-21. doi: 10.1002/1521-4141(200009)30:9<2713::AID-IMMU2713>3.0.CO;2-4.
4
Gallium disrupts iron metabolism of mycobacteria residing within human macrophages.镓会破坏人类巨噬细胞内分枝杆菌的铁代谢。
Infect Immun. 2000 Oct;68(10):5619-27. doi: 10.1128/IAI.68.10.5619-5627.2000.
5
CD1 and CD1-restricted T cells in infections with intracellular bacteria.细胞内细菌感染中的CD1及受CD1限制的T细胞
Trends Microbiol. 2000 Sep;8(9):419-25. doi: 10.1016/s0966-842x(00)01829-1.
6
Is the development of a new tuberculosis vaccine possible?开发一种新的结核病疫苗是否可行？
Nat Med. 2000 Sep;6(9):955-60. doi: 10.1038/79631.
7
Aerosol infection of mice with recombinant BCG secreting murine IFN-gamma partially reconstitutes local protective immunity.用分泌小鼠干扰素-γ的重组卡介苗对小鼠进行气溶胶感染，可部分重建局部保护性免疫。
Microb Pathog. 2000 Sep;29(3):175-85. doi: 10.1006/mpat.2000.0382.
8
Host immune response to intracellular bacteria: A role for MHC-linked class-Ib antigen-presenting molecules.
Proc Soc Exp Biol Med. 2000 Sep;224(4):231-9. doi: 10.1046/j.1525-1373.2000.22426.x.
9
Persistence of Mycobacterium tuberculosis in macrophages and mice requires the glyoxylate shunt enzyme isocitrate lyase.结核分枝杆菌在巨噬细胞和小鼠体内的持续存在需要乙醛酸循环酶异柠檬酸裂解酶。
Nature. 2000 Aug 17;406(6797):735-8. doi: 10.1038/35021074.
10
Mycobacterium tuberculosis infection in complement receptor 3-deficient mice.补体受体3缺陷小鼠中的结核分枝杆菌感染
J Immunol. 2000 Sep 1;165(5):2596-602. doi: 10.4049/jimmunol.165.5.2596.