实验性胰腺炎中甲状旁腺激素分泌增加与低钙血症：甲状腺完整的必要性。
Increased parathyroid hormone secretion and hypocalcemia in experimental pancreatitis: necessity for an intact thyroid gland.
作者信息
Norberg H P, DeRoos J, Kaplan E L
出版信息
Surgery. 1975 Jun;77(6):773-9.
Elevated concentrations of serum parathyroid hormone and hypocalcemia occur following the induction of experimental pancreatitis in the pig, but only when the thyroid gland is present. Prior thyroidectomy completely eliminates these changes. Serum magnesium concentrations remained normal throughout the experiments despite the occurrence of hypocalcemia. These data support the hypothesis that calcitonin or another thyroid-related substance plays a role in the mechanism of the hypocalcemia which accompanies experimental pancreatitis in the pig.
在猪身上诱导实验性胰腺炎后会出现血清甲状旁腺激素浓度升高和低钙血症，但仅在甲状腺存在时才会出现。事先进行甲状腺切除可完全消除这些变化。尽管出现了低钙血症，但在整个实验过程中血清镁浓度仍保持正常。这些数据支持这样一种假说，即降钙素或另一种与甲状腺相关的物质在猪实验性胰腺炎伴发的低钙血症机制中起作用。
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