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室性期外刺激诱发的与室上性心动过速相关的功能性束支阻滞消散的电生理特征。

Electrophysiologic characteristics of ventricular extrastimulation-induced dissipation of functional bundle branch block associated with supraventricular tachycardia.

作者信息

Wang S H, Han J, Hou C, Sung R J

机构信息

Department of Medicine, Stanford University School of Medicine, California, USA.

出版信息

J Cardiovasc Electrophysiol. 2001 Aug;12(8):928-34. doi: 10.1046/j.1540-8167.2001.00928.x.

Abstract

INTRODUCTION

Linking-related anterograde functional bundle branch block during supraventricular tachycardia (SVT) is due to repetitive concealed retrograde conduction of impulses from the contralateral bundle branch and can be eliminated by a critically timed premature ventricular beat (PVB). We assessed the electrophysiologic characteristics of PVB-induced dissipation of functional bundle branch block during SVT.

METHODS AND RESULTS

During SVT with functional bundle branch block, PVB was delivered from the right ventricular apex, scanning the tachycardia cycle length (CL) with 10-msec decrements in the coupling interval in 14 patients (3 AV nodal reentrant tachycardia and 11 orthodromic AV reciprocating tachycardia). Dissipation was achieved in group 1: functional right bundle branch block (RBBB) in 4, functional left bundle branch block (LBBB) in 4, and both functional RBBB and LBBB in 1 with a dissipation zone occupying 4% to 13% (mean 8.5%) of the tachycardia CL. The outer limits were 22+/-16 msec and 68+/-14 msec < tachycardia CL; the inner limits were 56+/-18 msec and 90+/-24 msec < tachycardia CL for RBBB and LBBB, respectively (both P < 0.05). Dissipation could not be achieved in group 2 (4 RBBB and 1 LBBB) due to CL-dependent bundle branch block and/or local ventricular refractoriness.

CONCLUSION

During SVT, functional bundle branch block due to "linking" often can be dissipated by timely PVB delivered from the right ventricular apex within a narrow zone of the tachycardia CL. Our findings suggest that the dissipation zone is affected by the pattern of functional bundle branch block relative to the site of PVB delivery.

摘要

引言

室上性心动过速(SVT)期间的连接相关顺行性功能性束支传导阻滞是由于冲动从对侧束支反复隐匿性逆行传导所致,可通过适时的室性早搏(PVB)消除。我们评估了PVB诱发的SVT期间功能性束支传导阻滞消散的电生理特征。

方法与结果

在伴有功能性束支传导阻滞的SVT期间,从右心室尖部发放PVB,在14例患者(3例房室结折返性心动过速和11例顺向性房室折返性心动过速)中,以10毫秒递减的偶联间期扫描心动过速周期长度(CL)。第1组实现了消散:4例功能性右束支传导阻滞(RBBB),4例功能性左束支传导阻滞(LBBB),1例同时存在功能性RBBB和LBBB,消散区占心动过速CL的4%至13%(平均8.5%)。RBBB的外部界限为心动过速CL < 22±16毫秒和68±14毫秒;LBBB的内部界限分别为心动过速CL < 56±18毫秒和90±24毫秒(均P < 0.05)。由于CL依赖性束支传导阻滞和/或局部心室不应期,第2组(4例RBBB和1例LBBB)未实现消散。

结论

在SVT期间,由“连接”引起的功能性束支传导阻滞通常可通过在心动过速CL的狭窄区内从右心室尖部适时发放PVB来消散。我们的研究结果表明,消散区受相对于PVB发放部位的功能性束支传导阻滞模式影响。

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