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半乳糖通过两种机制抑制生长素诱导的燕麦胚芽鞘生长。

Galactose inhibits auxin-induced growth of Avena coleoptiles by two mechanisms.

作者信息

Cheung S P, Cleland R E

机构信息

Department of Botany, University of Washington, Seattle 98195.

出版信息

Plant Cell Physiol. 1991;32(7):1015-9. doi: 10.1093/oxfordjournals.pcp.a078164.

Abstract

Galactose inhibits auxin-induced growth of Avena coleoptiles by at least two mechanisms. First, it inhibits auxin-induced H(+)-excretion needed for the initiation of rapid elongation. Galactose cannot be doing so by directly interfering with the ATPase since fusicoccin-induced H(+)-excretion is not affected. Secondly, galactose inhibits long-term auxin-induced growth, even in an acidic (pH 4.5) solution. This may be due to an inhibition of cell wall synthesis. However, galactose does not reduce the capacity of walls to be loosened by H+, given exogenously or excreted in response to fusicoccin.

摘要

半乳糖通过至少两种机制抑制生长素诱导的燕麦胚芽鞘生长。首先,它抑制快速伸长启动所需的生长素诱导的H⁺分泌。半乳糖不能通过直接干扰ATP酶来做到这一点,因为腐草霉素诱导的H⁺分泌不受影响。其次,半乳糖即使在酸性(pH 4.5)溶液中也会抑制生长素诱导的长期生长。这可能是由于细胞壁合成受到抑制。然而,半乳糖不会降低细胞壁被外源给予或因腐草霉素分泌的H⁺松弛的能力。

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