一种获取完整小鼠主动脉的方法，该方法可对动脉粥样硬化病变进行量化。 - Suppr

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超能文献

A procedure for obtaining whole mount mouse aortas that allows atherosclerotic lesions to be quantified.

作者信息

Brånén L, Pettersson L, Lindholm M, Zaina S

机构信息

Department of Medicine, University of Lund, Malmö General Hospital, Malmö, Sweden.

出版信息

Histochem J. 2001 Apr;33(4):227-9. doi: 10.1023/a:1017902509083.

DOI:10.1023/a:1017902509083

PMID:11550804

Abstract

摘要

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本文引用的文献

Monoclonal antibody against vascular cell adhesion molecule-1 inhibits neointimal formation after periadventitial carotid artery injury in genetically hypercholesterolemic mice.

Arterioscler Thromb Vasc Biol. 2000 Jul;20(7):1729-36. doi: 10.1161/01.atv.20.7.1729.

Changing concepts of atherogenesis.动脉粥样硬化形成的概念变迁

J Intern Med. 2000 Mar;247(3):349-58. doi: 10.1046/j.1365-2796.2000.00654.x.

The absence of p53 accelerates atherosclerosis by increasing cell proliferation in vivo.p53缺失通过增加体内细胞增殖加速动脉粥样硬化。

Nat Med. 1999 Mar;5(3):335-9. doi: 10.1038/6585.

Mouse models of angiogenesis, arterial stenosis, atherosclerosis and hemostasis.

Cardiovasc Res. 1998 Jul;39(1):8-33. doi: 10.1016/s0008-6363(98)00108-4.

State of the art: atherosclerosis in a limited edition.最新技术水平：限量版动脉粥样硬化。

Nat Med. 1998 Aug;4(8):899-900. doi: 10.1038/nm0898-899.

Quantitation of atherosclerosis in murine models: correlation between lesions in the aortic origin and in the entire aorta, and differences in the extent of lesions between sexes in LDL receptor-deficient and apolipoprotein E-deficient mice.小鼠模型中动脉粥样硬化的定量分析：主动脉起始处与整个主动脉病变之间的相关性，以及低密度脂蛋白受体缺陷型和载脂蛋白E缺陷型小鼠两性之间病变程度的差异。

J Lipid Res. 1995 Nov;36(11):2320-8.

Quantitative assessment of atherosclerotic lesions in mice.小鼠动脉粥样硬化病变的定量评估。

Atherosclerosis. 1987 Dec;68(3):231-40. doi: 10.1016/0021-9150(87)90202-4.

Spontaneous hypercholesterolemia and arterial lesions in mice lacking apolipoprotein E.缺乏载脂蛋白E的小鼠的自发性高胆固醇血症和动脉病变

Science. 1992 Oct 16;258(5081):468-71. doi: 10.1126/science.1411543.

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