Kraft L M, Vogel F S, Lloyd B, Benton E V, Cruty M R, Haymaker W, Leon A, Billingham J, Turnbill C E, Teas V, Look B C, Suri K, Miquel J, Ashley W W, Behnke A R, Samorajski T, Bailey O T, Zeman W
Aviat Space Environ Med. 1975 Apr;46(4 Sec 2):561-81.
The olfactory epithelium, but not the nasal respiratory epithelium, of the four pocket mice (Perognathus longimembris) that survived their flight on Apollo XVII showed both diffuse alterations and numerous disseminated focal lesions. The olfactory mucosa of the mouse that died during flight was also affected, but to a minor degree insofar as could be determined. All this was in contrast to the normal appearance of the olfactory mucosa of the numerous control animals. A number of possible causes were considered: systemic or regional infection; inhaled particulate material (seed dust); by-products from the KO2 bed in aerosol or particulate form; gas contaminants originating in the flight package; volatile substances from the dead mouse; weightlessness; and cosmic ray particle radiation. Where feasible, studies were conducted in an effort to rule in or rule out some of these potentially causative factors. No definitive conclusions were reached as to the cause of the lesions in the flight mice.
在阿波罗17号飞行任务中存活下来的四只囊鼠（长鼻囊鼠）的嗅觉上皮，而非鼻呼吸上皮，呈现出弥漫性改变和大量散在的局灶性病变。在飞行过程中死亡的囊鼠的嗅觉黏膜也受到了影响，但就所能确定的程度而言，影响较小。所有这些都与众多对照动物嗅觉黏膜的正常外观形成了对比。人们考虑了一些可能的原因：全身性或局部感染；吸入的颗粒物质（种子灰尘）；来自超氧化钾床的气溶胶或颗粒形式的副产品；飞行组件中产生的气体污染物；死鼠释放的挥发性物质；失重；以及宇宙射线粒子辐射。在可行的情况下，开展了研究以试图确定或排除其中一些潜在的致病因素。关于飞行囊鼠病变的原因尚未得出明确结论。
