Rizzon P, Rossi L, Baissus C, Demoulin J C, Di Biase M
Br Heart J. 1975 Jul;37(7):711-20. doi: 10.1136/hrt.37.7.711.
Fifteen cases of left posterior hemiblock associated with acute myocardial infaction were studied. In 5 cases the left posterior hemiblock was the only intraventricular conduction defect, while in the other 10 cases it was associated with complete right bundle-branch block. Left posterior hemiblock proved to be an early complication, appearing within a few hours from the onset of the acute episode, and an ominous sign, since hospital mortality rate was 87 per cent. Cause of death was mainly pump failure. In most of these cases ther was electrocardiographic evidence of infarction involving both anterior and inferior ventricular walls. Infarction of most or all of the ventricular septum was a common finding in the cases examined anatomically. Histologically, acute changes involving mainly the posterior septal and midseptal fibres were observed in 6 of the 8 cases studied. On the basis of these findings and of other published findings an alternative physiopathological mechanism for so-called left posterior hemiblock is proposed.
对15例与急性心肌梗死相关的左后分支阻滞病例进行了研究。5例中，左后分支阻滞是唯一的室内传导缺陷，而在其他10例中，它与完全性右束支阻滞相关。左后分支阻滞被证明是一种早期并发症，在急性发作开始后的数小时内出现，并且是一个不祥之兆，因为医院死亡率为87%。死亡原因主要是泵衰竭。在大多数这些病例中，心电图显示梗死累及心室前壁和下壁。在解剖检查的病例中，大部分或全部室间隔梗死是常见发现。组织学上，在研究的8例中的6例中观察到主要累及后间隔和中隔纤维的急性变化。基于这些发现以及其他已发表的发现，提出了所谓左后分支阻滞的另一种生理病理机制。
