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大肠杆菌细胞毒性坏死因子和博德特氏菌皮肤坏死毒素:激活Rho小GTP酶的皮肤坏死诱导毒素

Escherichia coli cytotoxic necrotizing factors and Bordetella dermonecrotic toxin: the dermonecrosis-inducing toxins activating Rho small GTPases.

作者信息

Horiguchi Y

机构信息

Project Research for Molecular Bacteriology, Research Institute for Microbial Diseases, Osaka University, Yamada-oka 3-1, Suita, 565-0871, Osaka, Japan.

出版信息

Toxicon. 2001 Nov;39(11):1619-27. doi: 10.1016/s0041-0101(01)00149-0.

Abstract

Escherichia coli cytotoxic necrotizing factors (CNFs) and Bordetella dermonecrotic toxin (DNT) have been recently found to comprise a novel family of dermonecrosis-inducing toxins which activate the small GTPases of the Rho family. They are single chain polypeptides consisting of an N-terminal domain responsible for binding to target cells and a C-terminal catalytic domain. CNFs (CNF1 and 2) and DNT share in the catalytic domain about 30% identical residues and a consensus sequence where the catalytically active center Cys resides. Both toxins deamidate Rho and other members of the Rho family, Rac and Cdc42, at Gln in the switch II region, which plays an important role in their GTPase activity. DNT, in addition, catalyzes a cross-link of the Gln of the GTPases with ubiquitous polyamines such as putrescine, spermidine, and spermine. The deamidation and the polyamination result in abrogation of the GTPase activity, and in addition, the polyamination endows Rho with the ability to interact with a downstream effector, ROCK, in a GTP-independent manner. These effects render the GTPases constitutively active, which underlies the toxicities of CNFs and DNT.

摘要

最近发现,大肠杆菌细胞毒性坏死因子(CNFs)和博德特氏菌皮肤坏死毒素(DNT)构成了一个新的诱导皮肤坏死毒素家族,它们可激活Rho家族的小GTP酶。它们是单链多肽,由负责与靶细胞结合的N端结构域和C端催化结构域组成。CNFs(CNF1和CNF2)和DNT在催化结构域中共有约30%的相同残基以及催化活性中心半胱氨酸所在的共有序列。两种毒素都在开关II区域的谷氨酰胺处使Rho以及Rho家族的其他成员Rac和Cdc42脱酰胺,这在它们的GTP酶活性中起重要作用。此外,DNT催化GTP酶的谷氨酰胺与诸如腐胺、亚精胺和精胺等普遍存在的多胺发生交联。脱酰胺和多胺化导致GTP酶活性丧失,此外,多胺化使Rho能够以不依赖GTP的方式与下游效应器ROCK相互作用。这些效应使GTP酶持续激活,这是CNFs和DNT毒性的基础。

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