Li L P, Buschmann M D, Shirazi-Adl A
Biosyntech Inc., Park of Science and High Technology, Laval, Canada.
J Biomech Eng. 2001 Oct;123(5):519-22. doi: 10.1115/1.1388295.
Observations in compression tests of articular cartilage have revealed unequal load increments for compression and release of the same amplitude applied to a disk with an identical previously imposed compression (in equilibrium). The mechanism of this asymmetric transient response is investigated here using a nonlinear fibril-reinforced model. It is found that the asymmetry is predominantly produced by the fibril stiffening with its tensile strain. In addition, allowing the hydraulic permeability to decrease significantly with compressive dilatation of cartilage increases the transient fibril strain, resulting in a stronger asymmetry. Large deformation also enhances the asymmetry as a consequence of stronger fibril stiffening.
对关节软骨压缩试验的观察表明，对于施加到具有相同先前施加压缩（处于平衡状态）的圆盘上的相同幅度的压缩和释放，载荷增量并不相等。本文使用非线性纤维增强模型研究了这种不对称瞬态响应的机制。研究发现，这种不对称主要是由纤维随其拉伸应变的硬化产生的。此外，随着软骨的压缩膨胀使液压渗透率显著降低，会增加瞬态纤维应变，从而导致更强的不对称性。由于更强的纤维硬化，大变形也会增强不对称性。
