Ritchey P N, Reid G S, Hasse L A
Department of Sociology, University of Cincinnati, Cincinnati, Ohio, USA.
J Adolesc Health. 2001 Dec;29(6):386-94. doi: 10.1016/s1054-139x(01)00260-9.
To construct and estimate a model that contains a reciprocal relationship between smoking and drinking and to test whether the gateway drug thesis or the cumulative risk behaviors thesis best fits the data.
Data (n = 630) are from a survey of all students (50% female; aged 16.2 years on average; 2.4 grade point average; and 57% residing in homes with both mother and father present) in a rural, tobacco-growing county's two high schools, one public (85%) and one private, in 1993. The survey was conducted by the schools as part of their alcohol and other drugs (AOD) prevention programs and was coordinated by the county AOD Council. Students completed the questionnaires in their homerooms. Endogenous predictors of drinking and smoking include student's perception of adult drug behavior, peer pressure to drink, degree to which their friends' drink, and attitudes toward drinking and smoking. Path coefficients were estimated by using LISREL.
The strong correlation between smoking and drinking resulted from shared causes, rather than from the effects of one type of drug use on the other. Approval of drinking had the strongest association with being a drinker (beta =.57) and with being a smoker (beta =.37). Those who found smoking offensive were less likely to be a smoker (beta = -.25). However, attitude toward smoking was not associated with being a drinker. Having drinking friends increased both the likelihood of being a drinker (beta =.29) and of being a smoker (beta =.23). Peer pressure to drink increased the likelihood of being a smoker (beta =.14) and of being a drinker (beta =.12). Students with lower grade point averages, males, older students, students in public school, and students with family structures other than both parents living in the same household were more likely to be a drinker and were more likely to be a smoker.
Our results support the cumulative risk behaviors thesis. The link between both high-risk behaviors, smoking and drinking, results from common causes rather than from drinking leading to smoking.
构建并评估一个包含吸烟与饮酒之间相互关系的模型,并检验通路药物理论或累积风险行为理论是否最符合数据。
数据(n = 630)来自1993年对一个种植烟草的农村县的两所高中所有学生(50%为女性;平均年龄16.2岁;平均绩点2.4;57%居住在父母都在家的家庭)的调查。该调查由学校作为其酒精及其他药物(AOD)预防项目的一部分进行,并由县AOD委员会协调。学生们在各自的教室完成问卷。饮酒和吸烟的内生预测因素包括学生对成人药物行为的认知、饮酒的同伴压力、朋友饮酒的程度以及对饮酒和吸烟的态度。路径系数通过使用LISREL进行估计。
吸烟与饮酒之间的强相关性源于共同原因,而非一种药物使用对另一种药物使用的影响。对饮酒的认可与成为饮酒者(β = 0.57)和吸烟者(β = 0.37)的关联最强。那些认为吸烟令人反感的人成为吸烟者的可能性较小(β = -0.25)。然而,对吸烟的态度与成为饮酒者无关。有饮酒的朋友增加了成为饮酒者(β = 0.29)和吸烟者(β = 0.23)的可能性。饮酒的同伴压力增加了成为吸烟者(β = 0.14)和饮酒者(β = 0.12)的可能性。平均绩点较低的学生、男性、年龄较大的学生、公立学校的学生以及家庭结构不是父母双方同住一个家庭的学生更有可能成为饮酒者,也更有可能成为吸烟者。
我们的结果支持累积风险行为理论。吸烟和饮酒这两种高风险行为之间的联系源于共同原因,而非饮酒导致吸烟。