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1
Ku86 is essential in human somatic cells.
Proc Natl Acad Sci U S A. 2002 Jan 22;99(2):832-7. doi: 10.1073/pnas.022649699. Epub 2002 Jan 15.
2
Heterozygous inactivation of human Ku70/Ku86 heterodimer does not affect cell growth, double-strand break repair, or genome integrity.
DNA Repair (Amst). 2006 Mar 7;5(3):303-11. doi: 10.1016/j.dnarep.2005.10.008. Epub 2005 Dec 1.
6
Translocation of Ku86/Ku70 to the multiple myeloma cell membrane: functional implications.
Exp Hematol. 2002 Mar;30(3):212-20. doi: 10.1016/s0301-472x(01)00786-x.
8
Regulation of telomere length and suppression of genomic instability in human somatic cells by Ku86.
Mol Cell Biol. 2004 Jun;24(11):5050-9. doi: 10.1128/MCB.24.11.5050-5059.2004.
9
Transfer of Ku86 RNA antisense decreases the radioresistance of human fibroblasts.
Cancer Gene Ther. 2000 Feb;7(2):339-46. doi: 10.1038/sj.cgt.7700111.
10
Ku86 represses lethal telomere deletion events in human somatic cells.
Proc Natl Acad Sci U S A. 2009 Jul 28;106(30):12430-5. doi: 10.1073/pnas.0903362106. Epub 2009 Jul 6.

引用本文的文献

1
Ku limits RNA-induced innate immunity to allow Alu expansion in primates.
Nature. 2025 Jul;643(8071):562-571. doi: 10.1038/s41586-025-09104-w. Epub 2025 May 15.
2
Targeting DNA damage sensors for cancer therapy.
DNA Repair (Amst). 2025 May;149:103841. doi: 10.1016/j.dnarep.2025.103841. Epub 2025 May 2.
3
Conversion of Ku80 K568 crotonylation to SUMOylation facilitates DNA non-homologous end joining and cancer radioresistance.
Signal Transduct Target Ther. 2025 Apr 21;10(1):127. doi: 10.1038/s41392-025-02210-1.
4
Growth hormone and radiation therapy: friend, foe, or both?
Endocr Relat Cancer. 2024 Jan 24;31(3). doi: 10.1530/ERC-22-0371. Print 2024 Mar 1.
5
Noncanonical functions of Ku may underlie essentiality in human cells.
Sci Rep. 2023 Jul 27;13(1):12162. doi: 10.1038/s41598-023-39166-7.
6
The APE2 nuclease is essential for DNA double-strand break repair by microhomology-mediated end joining.
Mol Cell. 2023 May 4;83(9):1429-1445.e8. doi: 10.1016/j.molcel.2023.03.017. Epub 2023 Apr 11.
7
Ku-DNA binding inhibitors modulate the DNA damage response in response to DNA double-strand breaks.
NAR Cancer. 2023 Feb 6;5(1):zcad003. doi: 10.1093/narcan/zcad003. eCollection 2023 Mar.
10
DNA End Joining: G0-ing to the Core.
Biomolecules. 2021 Oct 9;11(10):1487. doi: 10.3390/biom11101487.

本文引用的文献

2
Replication from oriP of Epstein-Barr virus requires human ORC and is inhibited by geminin.
Cell. 2001 Aug 10;106(3):287-96. doi: 10.1016/s0092-8674(01)00458-5.
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Non-homologous end joining as a mechanism of DNA repair.
Curr Biol. 2001 Jun 26;11(12):R455-7. doi: 10.1016/s0960-9822(01)00279-2.
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Securin is required for chromosomal stability in human cells.
Cell. 2001 May 18;105(4):445-57. doi: 10.1016/s0092-8674(01)00340-3.
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Post-translational modifications and activation of p53 by genotoxic stresses.
Eur J Biochem. 2001 May;268(10):2764-72. doi: 10.1046/j.1432-1327.2001.02225.x.
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Genome maintenance mechanisms for preventing cancer.
Nature. 2001 May 17;411(6835):366-74. doi: 10.1038/35077232.
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Cellular and biochemical impact of a mutation in DNA ligase IV conferring clinical radiosensitivity.
J Biol Chem. 2001 Aug 17;276(33):31124-32. doi: 10.1074/jbc.M103866200. Epub 2001 May 10.

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