未经治疗和已治疗的骨质疏松症中骨质流失和增加的机制。 - Suppr

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超能文献

Mechanisms of bone loss and gain in untreated and treated osteoporosis.

作者信息

Compston Juliet

机构信息

Department of Medicine, University of Cambridge School of Clinical Medicine, UK.

出版信息

Endocrine. 2002 Feb;17(1):21-7. doi: 10.1385/ENDO:17:1:21.

DOI:10.1385/ENDO:17:1:21

PMID:12014699

Abstract

摘要

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J Bone Miner Res. 2001 Jul;16(7):1300-7. doi: 10.1359/jbmr.2001.16.7.1300.

Effect of parathyroid hormone (1-34) on fractures and bone mineral density in postmenopausal women with osteoporosis.甲状旁腺激素（1-34）对绝经后骨质疏松症女性骨折及骨密度的影响。

N Engl J Med. 2001 May 10;344(19):1434-41. doi: 10.1056/NEJM200105103441904.

Effects of suppressed bone turnover by bisphosphonates on microdamage accumulation and biomechanical properties in clinically relevant skeletal sites in beagles.双膦酸盐抑制骨转换对比格犬临床相关骨骼部位微损伤积累和生物力学性能的影响。

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Alendronate increases bone strength by increasing the mean degree of mineralization of bone tissue in osteoporotic women.阿仑膦酸盐通过增加骨质疏松症女性骨组织的平均矿化程度来增强骨骼强度。

Bone. 2000 Nov;27(5):687-94. doi: 10.1016/s8756-3282(00)00376-8.

Histomorphometric evidence for increased bone turnover without change in cortical thickness or porosity after 2 years of cyclical hPTH(1-34) therapy in women with severe osteoporosis.在患有严重骨质疏松症的女性中，经两年周期性人甲状旁腺激素（1-34）[hPTH(1-34)]治疗后，骨转换增加但皮质厚度或骨孔隙率无变化的组织形态计量学证据。

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True strength.

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