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Initial proliferation of cortical cells in the formation of root nodules in Pisum sativum L.豌豆形成根瘤过程中皮层细胞的初始增殖。
Planta. 1973 Mar;114(1):17-28. doi: 10.1007/BF00390281.
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Nodulation and nitrogen fixation mutants of pea, Pisum sativum.豌豆结瘤和固氮突变体。
Theor Appl Genet. 1987 Oct;74(6):711-3. doi: 10.1007/BF00247546.
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Microscopic studies of cell divisions induced in alfalfa roots by Rhizobium meliloti.根瘤菌 meliloti 诱导苜蓿根细胞分裂的微观研究。
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Nodules Initiated by Rhizobium meliloti Exopolysaccharide Mutants Lack a Discrete, Persistent Nodule Meristem.根瘤菌胞外多糖突变体引发的根瘤缺乏离散的、持久的根瘤分生组织。
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Light Microscopy Study of Nodule Initiation in Pisum sativum L. cv Sparkle and in Its Low-Nodulating Mutant E2 (sym 5).豌豆 cv 火花结瘤起始的光镜研究及其低结瘤突变体 E2（sym5）
Plant Physiol. 1991 Nov;97(3):1206-11. doi: 10.1104/pp.97.3.1206.
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sym 13-A Gene Conditioning Ineffective Nodulation in Pisum sativum.sym13-A 基因调控豌豆结瘤无效。
Plant Physiol. 1990 Nov;94(3):899-905. doi: 10.1104/pp.94.3.899.
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Symbiotic Nitrogen Fixation.共生固氮作用
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8
Sym2 of Pea Is Involved in a Nodulation Factor-Perception Mechanism That Controls the Infection Process in the Epidermis.豌豆的Sym2参与一种根瘤因子感知机制，该机制控制表皮中的感染过程。
Plant Physiol. 1997 Oct;115(2):351-359. doi: 10.1104/pp.115.2.351.
9
Effect of mutations in Pisum sativum L. genes blocking different stages of nodule development on the expression of late symbiotic genes in Rhizobium leguminosarum bv. viciae.豌豆（Pisum sativum L.）中阻断根瘤发育不同阶段的基因突变对豌豆根瘤菌（Rhizobium leguminosarum bv. viciae）中晚期共生基因表达的影响。
Mol Plant Microbe Interact. 2001 Apr;14(4):471-6. doi: 10.1094/MPMI.2001.14.4.471.
10
The HCL gene of Medicago truncatula controls Rhizobium-induced root hair curling.蒺藜苜蓿的HCL基因控制根瘤菌诱导的根毛卷曲。
Development. 2001 May;128(9):1507-18. doi: 10.1242/dev.128.9.1507.

根瘤菌与豌豆（Pisum sativum L.）共生中感染过程起始和根瘤组织发育的遗传剖析。

Genetic dissection of the initiation of the infection process and nodule tissue development in the Rhizobium-pea (Pisum sativum L.) symbiosis.

作者信息

Tsyganov V E, Voroshilova V A, Priefer U B, Borisov A Y, Tikhonovich I A

机构信息

All-Russia Research Institute for Agricultural Microbiology, Laboratory of Genetics of Plant-Microbe Interactions, St-Petersburg, Pushkin.

出版信息

Ann Bot. 2002 Apr;89(4):357-66. doi: 10.1093/aob/mcf051.

DOI:10.1093/aob/mcf051
PMID:12096795
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4233866/
Abstract

Twelve non-nodulating pea (Pisum sativum L.) mutants were studied to identify the blocks in nodule tissue development. In nine, the reason for the lack of infection thread (IT) development was studied; this had been characterized previously in the other three mutants. With respect to IT development, mutants in gene sym7 are interrupted at the stage of colonization of the pocket in the curled root hair (Crh- phenotype), mutants in genes sym37 and sym38 are blocked at the stage of IT growth in the root hair cell (Ith- phenotype) and mutants in gene sym34 at the stage of IT growth inside root cortex cells (Itr- phenotype). With respect to nodule tissue development, mutants in genes sym7, sym14 and sym35 were shown to be blocked at the stage of cortical cell divisions (Ccd- phenotype), mutants in gene sym34 are halted at the stage of nodule primordium (NP) development (Npd- phenotype) and mutants in genes sym37 and sym38 are arrested at the stage of nodule meristem development (Nmd- phenotype). Thus, the sequential functioning of the genes Sym37, Sym38 and the gene Sym34 apparently differs in the infection process and during nodule tissue development. Based on these data, a scheme is suggested for the sequential functioning of early pea symbiotic genes in the two developmental processes: infection and nodule tissue formation.

摘要

研究了12个不结瘤豌豆（Pisum sativum L.）突变体，以确定根瘤组织发育中的阻断环节。对其中9个突变体研究了缺乏感染丝（IT）发育的原因；另外3个突变体的这一情况先前已有描述。关于IT发育，sym7基因的突变体在卷曲根毛（Crh-表型）中口袋状结构的定殖阶段被阻断，sym37和sym38基因的突变体在根毛细胞中IT生长阶段被阻断（Ith-表型），sym34基因的突变体在根皮层细胞内IT生长阶段被阻断（Itr-表型）。关于根瘤组织发育，sym7、sym14和sym35基因的突变体在皮层细胞分裂阶段被阻断（Ccd-表型），sym34基因的突变体在根瘤原基（NP）发育阶段停滞（Npd-表型），sym37和sym38基因的突变体在根瘤分生组织发育阶段停滞（Nmd-表型）。因此，Sym37、Sym38基因和Sym34基因在感染过程和根瘤组织发育过程中的顺序功能显然不同。基于这些数据，提出了一个早期豌豆共生基因在感染和根瘤组织形成这两个发育过程中顺序功能的示意图。