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本文引用的文献

1
Episodic coronary artery vasospasm and hypertension develop in the absence of Sur2 K(ATP) channels.在缺乏Sur2 K(ATP)通道的情况下，会出现发作性冠状动脉血管痉挛和高血压。
J Clin Invest. 2002 Jul;110(2):203-8. doi: 10.1172/JCI15672.
2
Mouse model of Prinzmetal angina by disruption of the inward rectifier Kir6.1.通过破坏内向整流钾离子通道蛋白6.1构建变异型心绞痛小鼠模型。
Nat Med. 2002 May;8(5):466-72. doi: 10.1038/nm0502-466.
3
Role of sarcolemmal K(ATP) channels in cardioprotection against ischemia/reperfusion injury in mice.肌膜ATP敏感性钾通道在小鼠心肌缺血/再灌注损伤心脏保护中的作用
J Clin Invest. 2002 Feb;109(4):509-16. doi: 10.1172/JCI14270.
4
Pharmacological comparison of native mitochondrial K(ATP) channels with molecularly defined surface K(ATP) channels.天然线粒体ATP敏感性钾通道与分子定义的表面ATP敏感性钾通道的药理学比较。
Mol Pharmacol. 2001 Feb;59(2):225-30.
5
Molecular basis of electrocardiographic ST-segment elevation.心电图ST段抬高的分子基础。
Circ Res. 2000 Nov 10;87(10):837-9. doi: 10.1161/01.res.87.10.837.
6
Molecular aspects of ATP-sensitive K+ channels in the cardiovascular system and K+ channel openers.心血管系统中ATP敏感性钾通道的分子层面及钾通道开放剂
Pharmacol Ther. 2000 Jan;85(1):39-53. doi: 10.1016/s0163-7258(99)00050-9.
7
ATP-sensitive K+ channels and insulin secretion: their role in health and disease.ATP敏感性钾通道与胰岛素分泌：它们在健康与疾病中的作用。
Diabetologia. 1999 Aug;42(8):903-19. doi: 10.1007/s001250051247.
8
Mitochondrial ATP-dependent potassium channels. Viable candidate effectors of ischemic preconditioning.线粒体ATP依赖性钾通道。缺血预处理的可行候选效应器。
Ann N Y Acad Sci. 1999 Jun 30;874:27-37. doi: 10.1111/j.1749-6632.1999.tb09222.x.
9
Association and stoichiometry of K(ATP) channel subunits.K(ATP)通道亚基的关联与化学计量学
Neuron. 1997 May;18(5):827-38. doi: 10.1016/s0896-6273(00)80321-9.
10
Inward rectifier potassium channels.内向整流钾通道
Annu Rev Physiol. 1997;59:171-91. doi: 10.1146/annurev.physiol.59.1.171.

The surprising role of vascular K(ATP) channels in vasospastic angina.

作者信息

Marbán Eduardo

机构信息

Institute of Molecular Cardiobiology, Johns Hopkins University, Baltimore, Maryland 21205, USA.

出版信息

J Clin Invest. 2002 Jul;110(2):153-4. doi: 10.1172/JCI16122.

DOI:10.1172/JCI16122
PMID:12122104
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC151068/
Abstract
摘要