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Gut oxygenation in sepsis: still a matter of controversy?脓毒症中的肠道氧合：仍是一个有争议的问题？
Crit Care. 2002 Aug;6(4):282-3. doi: 10.1186/cc1508. Epub 2002 May 31.
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Effects of hemodilution and oxygen breathing on gut oxygenation in anesthetized dogs.血液稀释和吸氧对麻醉犬肠道氧合的影响。
Adv Exp Med Biol. 1997;411:323-9. doi: 10.1007/978-1-4615-5865-1_40.
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Mechanism of hepatocellular dysfunction during sepsis: the role of gut-derived norepinephrine (review).脓毒症期间肝细胞功能障碍的机制：肠道源性去甲肾上腺素的作用（综述）
Int J Mol Med. 2000 May;5(5):457-65. doi: 10.3892/ijmm.5.5.457.
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Effect of hypoxic hypoxia on transmural gut and subcutaneous tissue oxygen tension.低氧性缺氧对跨壁肠组织和皮下组织氧张力的影响。
Adv Exp Med Biol. 1997;411:319-22. doi: 10.1007/978-1-4615-5865-1_39.
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Early gut ischemia in experimental fecal peritonitis.实验性粪性腹膜炎中的早期肠道缺血
Circ Shock. 1992 Sep;38(1):22-8.
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Cellular aspects of shock, sepsis and burns: mechanisms of cell injury.休克、脓毒症和烧伤的细胞层面：细胞损伤机制
Prog Clin Biol Res. 1988;264:3-6.
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Radical explanations for old observations.对旧有观察结果的根本性解释。
Gastroenterology. 1987 Jun;92(6):2026-8. doi: 10.1016/0016-5085(87)90640-8.
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Assessment of adequate tissue oxygenation in shock and critical illness: oxygen transport in sepsis, Bermuda, April 1 + 2, 1989.评估休克和危重病中组织氧合是否充足：脓毒症中的氧输送，百慕大，1989年4月1日至2日
Intensive Care Med. 1989;15(7):475-7. doi: 10.1007/BF00255606.
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Tissue oxygenation in sepsis; new insights from in vivo EPR.脓毒症中的组织氧合；体内电子顺磁共振的新见解
NMR Biomed. 2004 Aug;17(5):319-26. doi: 10.1002/nbm.910.
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Oxygen delivery to tissues.氧气向组织的输送。
Eur Respir J. 1991 Nov;4(10):1258-67.
引用本文的文献
1
Increased blood flow prevents intramucosal acidosis in sheep endotoxemia: a controlled study.增加血流量可预防绵羊内毒素血症时的黏膜内酸中毒：一项对照研究。
Crit Care. 2005 Apr;9(2):R66-73. doi: 10.1186/cc3021. Epub 2005 Jan 11.
本文引用的文献
1
Clinical review: splanchnic ischaemia.临床综述：内脏缺血
Crit Care. 2002 Aug;6(4):306-12. doi: 10.1186/cc1515. Epub 2002 Apr 8.
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Cytopathic hypoxia. Is oxygen use impaired in sepsis as a result of an acquired intrinsic derangement in cellular respiration?细胞病变性缺氧。败血症时，细胞呼吸出现后天性内在紊乱，是否会导致氧利用受损？
Crit Care Clin. 2002 Jan;18(1):165-75. doi: 10.1016/s0749-0704(03)00071-x.
3
Early goal-directed therapy in the treatment of severe sepsis and septic shock.早期目标导向治疗在严重脓毒症和脓毒性休克治疗中的应用
N Engl J Med. 2001 Nov 8;345(19):1368-77. doi: 10.1056/NEJMoa010307.
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Increased ileal-mucosal-arterial PCO2 gap is associated with impaired villus microcirculation in endotoxic pigs.回肠黏膜动脉血二氧化碳分压差值升高与内毒素血症猪的绒毛微循环受损有关。
Intensive Care Med. 2001 Apr;27(4):757-66. doi: 10.1007/s001340100871.
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Endotoxin-induced ileal mucosal injury and nitric oxide dysregulation are temporally dissociated.内毒素诱导的回肠黏膜损伤与一氧化氮调节异常在时间上是分离的。
Am J Respir Crit Care Med. 2000 May;161(5):1705-12. doi: 10.1164/ajrccm.161.5.9907043.
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Abnormalities of gastric mucosal oxygenation in septic shock: partial responsiveness to dopexamine.脓毒性休克时胃黏膜氧合异常：对多培沙明的部分反应性。
Am J Respir Crit Care Med. 1998 May;157(5 Pt 1):1586-92. doi: 10.1164/ajrccm.157.5.9710017.
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Effects of short-term endotoxemia and dopamine on mucosal oxygenation in porcine jejunum.短期内毒素血症和多巴胺对猪空肠黏膜氧合的影响。
Am J Physiol. 1996 Apr;270(4 Pt 1):G667-75. doi: 10.1152/ajpgi.1996.270.4.G667.
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Gut and muscle tissue PO2 in endotoxemic dogs during shock and resuscitation.内毒素血症犬在休克和复苏过程中肠道和肌肉组织的氧分压
J Appl Physiol (1985). 1994 Feb;76(2):793-800. doi: 10.1152/jappl.1994.76.2.793.
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Nitric oxide dilates tight junctions and depletes ATP in cultured Caco-2BBe intestinal epithelial monolayers.一氧化氮可使培养的Caco-2BBe肠上皮单层细胞中的紧密连接扩张并消耗三磷酸腺苷（ATP）。
Am J Physiol. 1995 Feb;268(2 Pt 1):G361-73. doi: 10.1152/ajpgi.1995.268.2.G361.
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Nanomolar concentrations of nitric oxide reversibly inhibit synaptosomal respiration by competing with oxygen at cytochrome oxidase.纳摩尔浓度的一氧化氮通过在细胞色素氧化酶处与氧竞争，可逆地抑制突触体呼吸。
FEBS Lett. 1994 Dec 19;356(2-3):295-8. doi: 10.1016/0014-5793(94)01290-3.
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