Nachum Zohar, Ben-Shlomo Izhar, Rakover Yardena, Weiner Ehud, Shalev Eliezer
Department of Obstetrics and Gynecology, The Pediatric Endocrine Unit and Endocrine Laboratory, HaEmek Medical Center, Afula, Israel.
J Pediatr. 2002 Nov;141(5):706-9. doi: 10.1067/mpd.2002.128546.
We present two siblings with persistent hyperinsulinemic hypoglycemia of infancy, accelerated intrauterine growth and early neonatal brain atrophy. Fetal plasma glucose and insulin levels in the second sibling revealed normoglycemia despite hyperinsulinemia. The absence of intrauterine hypoglycemia suggests that the brain damage is not secondary to hypoglycemia and other etiologies must be considered.
我们报告了两名患有持续性婴儿高胰岛素血症性低血糖症、宫内生长加速和早期新生儿脑萎缩的兄弟姐妹。第二名兄弟姐妹的胎儿血浆葡萄糖和胰岛素水平显示，尽管存在高胰岛素血症，但血糖正常。宫内无低血糖表明脑损伤并非继发于低血糖，必须考虑其他病因。
