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The molecular events underlying ischemia/reperfusion injury.

作者信息

Anaya-Prado R, Toledo-Pereyra L H

机构信息

Borgess Research Institute, Trauma, Surgery Research Sciences and Molecular Biology, Kalamazoo, Michigan 49048, USA.

出版信息

Transplant Proc. 2002 Nov;34(7):2518-9. doi: 10.1016/s0041-1345(02)03471-1.

DOI:10.1016/s0041-1345(02)03471-1
PMID:12431508
Abstract
摘要

相似文献

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The molecular events underlying ischemia/reperfusion injury.
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Leukocytes and endothelium interaction as rate limiting step in the inflammatory response and a key factor in the ischemia-reperfusion injury.白细胞与内皮细胞的相互作用是炎症反应中的限速步骤以及缺血再灌注损伤的关键因素。
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Soluble ICAM-1 reduces leukocyte adhesion to vascular endothelium in ischemia-reperfusion injury in mice.可溶性细胞间黏附分子-1可减少小鼠缺血再灌注损伤中白细胞与血管内皮的黏附。
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Behring Inst Mitt. 1993 Aug(92):229-37.

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