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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Thompson S H, Smith S J

J Neurophysiol. 1976 Jan;39(1):153-61. doi: 10.1152/jn.1976.39.1.153.

Spikes in molluscan bursting cells are followed by depolarizing afterpotentials (DAPs) which are not seen in nonbursting cells in the same ganglia. DAPs from successive spikes sum to provide a depolarizing drive capable of sustaining multiple discharge. 2. Subthreshold depolarization activates a DAP-like process in bursters. 3. DAP amplitude increases as the cell is hyperpolarized beyond the potassium equilibrium potential. The amplitude is not changed by intracellular iontophoresis of TEA-Cl. DAP amplitude is reduced by 39% after a 10-min exposure to CA++-free saline, and by 66% after 10 min in 4% Na+ (Tris substituted) saline. The amplitude is unchanged by exposure to K+-free saline. 4. During repetitive stimulation the amplitude of the summed DAP declines. 5. It is concluded that DAP results from a slowly decaying component of Ca++ and Na+ permeability.

软体动物爆发性细胞的动作电位之后会出现去极化后电位（DAPs），而同一神经节中的非爆发性细胞则不会出现这种情况。连续动作电位产生的DAPs会叠加，从而提供一种能够维持多次放电的去极化驱动力。2. 阈下去极化会激活爆发性细胞中的类似DAP的过程。3. 当细胞超极化超过钾离子平衡电位时，DAP的幅度会增加。TEA-Cl的细胞内离子导入不会改变该幅度。在无钙盐溶液中暴露10分钟后，DAP幅度降低39%，在4% Na+（三羟甲基氨基甲烷替代）盐溶液中暴露10分钟后，DAP幅度降低66%。暴露于无钾盐溶液中时，幅度不变。4. 在重复刺激期间，叠加的DAP的幅度会下降。5. 得出的结论是，DAP是由Ca++和Na+通透性的缓慢衰减成分引起的。

Thompson S H, Smith S J

J Neurophysiol. 1976 Jan;39(1):153-61. doi: 10.1152/jn.1976.39.1.153.

Spikes in molluscan bursting cells are followed by depolarizing afterpotentials (DAPs) which are not seen in nonbursting cells in the same ganglia. DAPs from successive spikes sum to provide a depolarizing drive capable of sustaining multiple discharge. 2. Subthreshold depolarization activates a DAP-like process in bursters. 3. DAP amplitude increases as the cell is hyperpolarized beyond the potassium equilibrium potential. The amplitude is not changed by intracellular iontophoresis of TEA-Cl. DAP amplitude is reduced by 39% after a 10-min exposure to CA++-free saline, and by 66% after 10 min in 4% Na+ (Tris substituted) saline. The amplitude is unchanged by exposure to K+-free saline. 4. During repetitive stimulation the amplitude of the summed DAP declines. 5. It is concluded that DAP results from a slowly decaying component of Ca++ and Na+ permeability.

软体动物爆发性细胞的动作电位之后会出现去极化后电位（DAPs），而同一神经节中的非爆发性细胞则不会出现这种情况。连续动作电位产生的DAPs会叠加，从而提供一种能够维持多次放电的去极化驱动力。2. 阈下去极化会激活爆发性细胞中的类似DAP的过程。3. 当细胞超极化超过钾离子平衡电位时，DAP的幅度会增加。TEA-Cl的细胞内离子导入不会改变该幅度。在无钙盐溶液中暴露10分钟后，DAP幅度降低39%，在4% Na+（三羟甲基氨基甲烷替代）盐溶液中暴露10分钟后，DAP幅度降低66%。暴露于无钾盐溶液中时，幅度不变。4. 在重复刺激期间，叠加的DAP的幅度会下降。5. 得出的结论是，DAP是由Ca++和Na+通透性的缓慢衰减成分引起的。