葡萄糖-6-磷酸脱氢酶缺乏的同胞中的急性可逆性肾衰竭。
Acute reversible renal failure in G-6 PD-deficient siblings.
作者信息
Gulati P D, Rizvi S N
出版信息
Postgrad Med J. 1976 Feb;52(604):83-5. doi: 10.1136/pgmj.52.604.83.
Abstract
Two G-6 PD-deficient siblings, who initially presented with intravascular haemolysis, and later developed acute oliguric renal failure, are described. Renal failure reversed as haemolysis regressed in both patients. The pathogenesis of this complication is reported to be multifactorial but in our patients tubular obstruction by haemoglobin casts is the most probable mechanism. The importance of recognizing this renal complication in G-6 PD-deficient subjects is stressed.
摘要
本文描述了两名葡萄糖-6-磷酸脱氢酶（G-6 PD）缺乏的同胞，他们最初表现为血管内溶血，随后发展为急性少尿性肾衰竭。随着溶血在两名患者中消退，肾衰竭也得到逆转。据报道，这种并发症的发病机制是多因素的，但在我们的患者中，血红蛋白管型导致的肾小管阻塞是最可能的机制。文中强调了在G-6 PD缺乏的受试者中识别这种肾脏并发症的重要性。
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