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[使用双十极电极导管和CARTO系统进行联合标测以诊断伴有周长交替的切口折返性房性心动过速的可行性:病例报告]

[Feasibility of combination mapping utilizing a duo-decapolar electrode catheter and the CARTO system for incisional reentrant atrial tachycardia with cycle length alternation: a case report].

作者信息

Sohara Hiroshi, Tanaka Kazushi, Ishigaki Kiyoko, Yamaguchi Yoshio, Murakami Masato, Takahashi Saeko, Taketani Yoshio, Sunami Kazuhiro, Miyashita Yusuke, Hiroe Yoshitaka, Tanaka Shinji, Saito Shigeru, Satake Shutaro, Watanabe Yoshio

机构信息

Division of Cardiology, Heart Center, Shonan Kamakura General Hospital, Yamazaki 1202-1, Kamakura, Kanagawa 247-8533.

出版信息

J Cardiol. 2003 Mar;41(3):135-42.

Abstract

A 43-year-old woman had undergone patch closure operation for atrial septal defect 27 years ago. She was referred to our hospital for evaluation of frequent palpitations since 1 year ago. Electrophysiological study was performed with recording of the coronary sinus, His bundle, and low lateral right free wall electrography utilizing a steerable duo-decapolar electrode catheter(Livewire, Daig). Supraventricular tachycardia with cycle length alternation of 300 and 320 msec similar to atrial flutter was reproducibly provoked by burst pacing from the coronary sinus. During the supraventricular tachycardia, abnormal atrial potentials occurred in the low lateral right free wall region with very low amplitude and splitting potentials. The cycle length alternation of the supraventricular tachycardia depended on the occurrence of the splitting potentials, that is, the splitting potentials were present during the supraventricular tachycardia with a long cycle and the splitting potentials were absent during the supraventricular tachycardia with a short cycle. This phenomenon suggested that the splitting potentials resulted from 2:1 functional intra-atrial local conduction block. In addition, during sinus rhythm the abnormal electrograms revealed fractionated activity. Thus, these findings strongly imply that the supraventricular tachycardia is due to a macro-reentrant right atrial tachycardia utilizing an anatomical obstacle caused by the atrial septal defect operation as a central area, namely incisional reentrant atrial tachycardia. Three-dimensional electroanatomical mapping using the CARTO system(Biosense-Webster) was conducted to investigate whether the low lateral right free wall area possessed the critical isthmus essential to the reentry circuit. Electroanatomical mapping revealed that the very low amplitude potentials and the splitting potentials corresponded to the scars and the functional conduction block area detected by mapping using the multipolar catheter, respectively. According to the propagation mapping, the incisional reentrant atrial tachycardia slowly conducted the channel created by multiple neighboring scars clockwise and the alternation of the tachycardia cycle length was dependent on the development of the functional local intra-atrial conduction block within the channel. An approximately 1.5 cm successful linear lesion was created by radiofrequency catheter ablation to transect the isthmus based on the electroanatomical mapping findings. Afterwards, the incisional reentrant atrial tachycardia could not be induced by burst stimuli from the coronary sinus even under administration of isoproterenol. The use of three dimensional electroanatomical mapping(CARTO system) to evaluate the reentry circuit after the detection of abnormal potentials by using multipolar catheter in advance is a very useful method to determine optimal target site of ablation for a patient with incisional reentrant atrial tachycardia.

摘要

一名43岁女性27年前因房间隔缺损接受了补片闭合手术。自1年前起,她因频繁心悸被转诊至我院。使用可操控的双十极电极导管(Livewire,Daig)进行电生理研究,记录冠状窦、希氏束和右心室游离壁外侧低位电图。通过冠状窦猝发起搏可重复性诱发与心房扑动相似的室上性心动过速,其周期长度交替为300和320毫秒。在室上性心动过速期间,右心室游离壁外侧低位区域出现异常心房电位,振幅极低且电位分裂。室上性心动过速的周期长度交替取决于电位分裂的出现,即长周期的室上性心动过速期间存在电位分裂,短周期的室上性心动过速期间不存在电位分裂。这一现象提示电位分裂是由2:1功能性心房内局部传导阻滞所致。此外,在窦性心律时,异常电图显示出碎裂活动。因此,这些发现强烈提示室上性心动过速是由于以房间隔缺损手术造成的解剖学障碍为中心区域的大折返性右房心动过速,即切口折返性房性心动过速。使用CARTO系统(Biosense-Webster)进行三维电解剖标测,以研究右心室游离壁外侧低位区域是否存在折返环路所必需的关键峡部。电解剖标测显示,极低振幅电位和电位分裂分别对应于使用多极导管标测检测到的瘢痕和功能性传导阻滞区域。根据激动标测,切口折返性房性心动过速沿多个相邻瘢痕形成的通道顺时针缓慢传导,心动过速周期长度的交替取决于通道内功能性局部心房内传导阻滞的发展。基于电解剖标测结果,通过射频导管消融创建了一条约1.5厘米的成功线性病变以横断峡部。此后,即使在使用异丙肾上腺素的情况下,冠状窦猝发起搏也无法诱发切口折返性房性心动过速。对于切口折返性房性心动过速患者,在预先使用多极导管检测到异常电位后,使用三维电解剖标测(CARTO系统)评估折返环路是确定最佳消融靶点的非常有用的方法。

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