Oshiro Shinya, Fukushima Takeo
Department of Neurosurgery, Okinawa Prefectural Miyako Hospital, 807 Higashi-Nakasone, Hirara, Okinawa 906-0007, Japan.
No To Shinkei. 2003 Mar;55(3):265-8.
We report a 74-year-old man with an ischemic lesion in the ventral midbrain. He presented with contralateral ptosis and marked upward gaze paresis of the right eye. Neurological examination revealed partial oculomotor nerve palsy caused by impairment of the right levator palpebrae, superior rectus and inferior oblique muscles. This finding is highly suggestive of a possible lesion in the midbrain affecting the oculomotor fascicular fibers. Magnetic resonance images showed an ischemic lesion in the paramedian area of the right midbrain tegmentum. The coronal view of T 2-weighted imaging clearly demonstrated to be the site of lesions below the red nucleus. It seemed to be coincidental with the impaired site of involving the caudal part of oculomotor fascicular fibers emerging from the nucleus. This report is considered to be a typical case of partial fascicular oculomotor paresis based on impairment of the caudal part of oculomotor fascicles for the levator palpebrae, superior rectus, and inferior oblique muscles. This is a valuable case to be documented in which neurological site of lesions are consistent with those found in radiological study.
我们报告一名74岁男性,其腹侧中脑存在缺血性病变。他表现为对侧上睑下垂及右眼明显的上视麻痹。神经系统检查发现右侧提上睑肌、上直肌和下斜肌受损导致部分动眼神经麻痹。这一发现高度提示中脑可能存在影响动眼神经束状纤维的病变。磁共振成像显示右侧中脑被盖部旁正中区域存在缺血性病变。T2加权成像的冠状位视图清楚地显示病变部位在红核下方。这似乎与动眼神经核发出的动眼神经束状纤维尾端受损部位相吻合。本报告被认为是基于动眼神经束状纤维尾端对提上睑肌、上直肌和下斜肌的损害而导致部分束状动眼神经麻痹的典型病例。这是一个有价值的病例记录,其中神经病变部位与放射学研究结果一致。