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房室结通路的退行性病变导致二度和三度房室传导阻滞。

A degenerative lesion of the approach to the atrioventricular node producing second-degree and third-degree atrioventricular block.

作者信息

Kawai S, Fu L, Aziki K, Okada R, Katoh K

机构信息

Research Laboratory for Cardiovascular Pathology, Juntendo University, Tokyo, Japan.

出版信息

Pacing Clin Electrophysiol. 1992 Dec;15(12):2263-9. doi: 10.1111/j.1540-8159.1992.tb04170.x.

DOI:10.1111/j.1540-8159.1992.tb04170.x
PMID:1282248
Abstract

We report a case of a degenerative approach lesion in an 83-year-old male with diabetes mellitus, hypertension, and ischemic heart disease. His ECGs changed from first-degree atrioventricular (AV) block 14 years ago, to third-degree AV (A-H) block. A pacemaker was implanted for bradycardia. He died 4 years later from heart and renal failure. Serial sections through the conduction system revealed total depletion and fatty replacement of the atrial muscle at the approaches to the AV node.

摘要

我们报告一例83岁男性患者,患有糖尿病、高血压和缺血性心脏病,存在退行性传导系统病变。他的心电图在14年前为一度房室传导阻滞,后来发展为三度房室(A-H)传导阻滞。因心动过缓植入了起搏器。4年后,他死于心力衰竭和肾衰竭。对传导系统进行连续切片检查发现,房室结附近的心房肌完全缺失并被脂肪替代。

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A degenerative lesion of the approach to the atrioventricular node producing second-degree and third-degree atrioventricular block.房室结通路的退行性病变导致二度和三度房室传导阻滞。
Pacing Clin Electrophysiol. 1992 Dec;15(12):2263-9. doi: 10.1111/j.1540-8159.1992.tb04170.x.
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