Diagana M, Khalil M, Preux P M, Dumas M, Jauberteau M O
L'Institut d'Epidémiologie Neurologique et de Neurologie Tropicale, Centre Hospitalier Universitaire, Limoges, France.
Med Trop (Mars). 2003;63(1):68-74.
Several explanations have been proposed to explain the relationship between axonal forms of acute auto-immune inflammatory polyradiculoneuritis and Campylobacter jejuni. The major hypothesis involving molecular imitation is based on the existence of common antigenic determinants (epitopes) in the lipopolysaccharides of the infectious agent and gangliosides, i.e. glycosphingolipides on the surface of the nervous system cells, especially peripheral nervous system cells. The purpose of this literature review is to improve understanding of the rather complex physiopathological mechanisms underlying Guillain-Barre syndrome.
已经提出了几种解释来阐述急性自身免疫性炎性多发性神经根神经炎的轴突形式与空肠弯曲菌之间的关系。涉及分子模拟的主要假说是基于感染因子的脂多糖与神经节苷脂（即神经系统细胞尤其是外周神经系统细胞表面的糖鞘脂）中存在共同的抗原决定簇（表位）。这篇文献综述的目的是增进对吉兰 - 巴雷综合征潜在的相当复杂的生理病理机制的理解。
