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[某些实验性缺损中心脏和肺部的血管]

[Vessels of the heart and lung in some experimental defects].

作者信息

Gibradze T A

出版信息

Arkh Anat Gistol Embriol. 1975 Nov;69(11):14-9.

PMID:129046
Abstract

Under study were changes of intraorganic blood vessels of the heart and lungs in some experimental defects (open arterial defect, coarctation of the aorta, simultaneous existence of these two defects, stenosis of the pulmonary trunk, defect of the interatrial septum, triad of Fallot, syndrom of Lutembachet). Morphological data correlated with blood pressure in the pulmonary circulation and cardiac chambers. The complex of compensatory-adaptational mechanisms consisting of comparatively active and passive zones is formed in the heart and lungs. In most cases the changes develop in the vessels already existing. In hypertrophy of the myocardium when there is hypertension and hypervolemia in coronary vessels, sinusoids perform the function of blood reservoir, to a certain degree balancing the blood pressure, and luminar ducts relieve the muscle from excessive blood. The changes in the vascular system of the lung are directly dependent upon the pressure in the pulmonary circulation and the duration of observation. The closing arteries are the most active link in the chain of compensatory-adaptational mechanisms.

摘要

本研究观察了某些实验性缺损(开放性动脉缺损、主动脉缩窄、这两种缺损同时存在、肺动脉干狭窄、房间隔缺损、法洛三联症、鲁登巴赫综合征)中心脏和肺部器官内血管的变化。形态学数据与肺循环和心腔内的血压相关。心脏和肺部形成了由相对活跃和被动区域组成的代偿 - 适应机制复合体。在大多数情况下,变化发生在已有的血管中。在心肌肥厚伴有冠状动脉高血压和血容量过多时,血窦起到血液储存库的作用,在一定程度上平衡血压,而管腔管道则使心肌免受过多血液的影响。肺部血管系统的变化直接取决于肺循环压力和观察持续时间。闭锁动脉是代偿 - 适应机制链中最活跃的环节。

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