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在一名患有活化蛋白C抵抗的患者中偶然发现硬脑膜动静脉瘘。

Incidental discovery of a dural arteriovenous fistula in a patient with activated protein C resistance.

作者信息

Wenderoth Jason D, Phatouros Constantine C

机构信息

Interventional Neuroradiology Unit, Royal Perth Hospital, Perth, WA, Australia.

出版信息

AJNR Am J Neuroradiol. 2003 Aug;24(7):1369-71.

PMID:12917129
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7973649/
Abstract

A dural arteriovenous fistula was discovered incidentally in a 58-year-old man with activated protein C resistance who underwent routine outpatient cerebral angiography for workup of multiple intracranial aneurysms.

摘要

一名58岁患有活化蛋白C抵抗的男性在因多发性颅内动脉瘤进行常规门诊脑血管造影检查时,偶然发现了硬脑膜动静脉瘘。

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引用本文的文献

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本文引用的文献

1
Molecular analysis of thrombophilic risk factors in patients with dural arteriovenous fistulas.硬脑膜动静脉瘘患者血栓形成风险因素的分子分析
J Neurol. 2002 Jun;249(6):680-2. doi: 10.1007/s00415-002-0690-8.
2
Multidisciplinary management of spinal dural arteriovenous fistulas: clinical presentation and long-term follow-up in 49 patients.脊髓硬脊膜动静脉瘘的多学科管理:49例患者的临床表现及长期随访
Stroke. 2002 Jun;33(6):1578-83. doi: 10.1161/01.str.0000018009.83713.06.
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Clinical characteristics of dural arteriovenous fistula.硬脑膜动静脉瘘的临床特征
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Significantly increased prevalence of factor V Leiden in patients with dural arteriovenous fistulas.
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Association of resistance to activated protein C and dural arteriovenous fistulas.活化蛋白C抵抗与硬脑膜动静脉瘘的关联
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Resistance to activated protein C caused by the R506Q mutation in the gene for factor V is a common risk factor for venous thrombosis.因子V基因中R506Q突变导致的活化蛋白C抵抗是静脉血栓形成的常见危险因素。
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Cerebral venous thrombosis and activated protein C resistance.
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