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Intermittent aldosteronism in periodic paralysis; dependence of attacks on retention of sodium, and failure to induce attacks by restriction of dietary sodium.

作者信息

CONN J W, FAJANS S S, LOUIS L H, STREETEN D H, JOHNSON R D

出版信息

Lancet. 1957 Apr 20;272(6973):802-5. doi: 10.1016/s0140-6736(57)90970-4.

DOI:10.1016/s0140-6736(57)90970-4
PMID:13417599
Abstract
摘要

相似文献

1
Intermittent aldosteronism in periodic paralysis; dependence of attacks on retention of sodium, and failure to induce attacks by restriction of dietary sodium.周期性麻痹中的间歇性醛固酮增多症；发作对钠潴留的依赖性，以及通过限制饮食中的钠来诱发发作的失败。
Lancet. 1957 Apr 20;272(6973):802-5. doi: 10.1016/s0140-6736(57)90970-4.
2
Periodic paralysis; sodium metabolism and aldosterone output in two cases.周期性麻痹；两例患者的钠代谢与醛固酮分泌
Lancet. 1959 Jan 24;1(7065):177-81. doi: 10.1016/s0140-6736(59)90371-x.
3
Dependence of attacks of periodic paralysis upon retention of sodium; and failure to induce either paralysis or sequestration of potassium when dietary sodium is restricted.周期性麻痹发作对钠潴留的依赖性；以及当饮食中钠受限，无法诱发麻痹或钾潴留。
Trans Assoc Am Physicians. 1957;70:167-82.
4
Primary aldosteronism and salt.原发性醛固酮增多症与盐
Pflugers Arch. 2015 Mar;467(3):587-94. doi: 10.1007/s00424-014-1658-0. Epub 2014 Dec 13.
5
Renal tubular acidosis. The role of defective renal tubular sodium reabsorption and secondary hyperaldosteronism in its pathogenesis.
Arch Intern Med. 1959 Oct;104:613-8.
6
The electrocardiogram and potassium metabolism; electrocardiographic abnormalities in primary aldosteronism and familial periodic paralysis.心电图与钾代谢；原发性醛固酮增多症和家族性周期性麻痹中的心电图异常
Am J Med. 1957 Sep;23(3):376-84. doi: 10.1016/0002-9343(57)90317-0.
7
The phosphorylated sodium chloride cotransporter in urinary exosomes is superior to prostasin as a marker for aldosteronism.尿外泌体中磷酸化的氯化钠协同转运蛋白优于前列腺素作为醛固酮增多症的标志物。
Hypertension. 2012 Sep;60(3):741-8. doi: 10.1161/HYPERTENSIONAHA.112.198135. Epub 2012 Jul 30.
8
Useful parameters in the diagnosis of primary aldosteronism.原发性醛固酮增多症诊断中的有用参数。
JAMA. 1961 Oct 7;178:19-22. doi: 10.1001/jama.1961.03040400021004.
9
Observations on a presumptive case of primary aldosteronism.
J Lab Clin Med. 1956 Jul;48(1):1-12.
10
Aldosterone revisited.再谈醛固酮。
N Engl J Med. 2004 Nov 11;351(20):2131-3; author reply 2131-3.

引用本文的文献

1
Hyperthyroidism Complicated by Periodic Paralysis.甲状腺功能亢进症合并周期性麻痹
Postgrad Med J. 1965 May;41(475):286-7. doi: 10.1136/pgmj.41.475.286.
2
[ON THE ELECTROLYTE AND ALDOSTERONE METABOLISM IN ADYNAMIA EPISODICA HEREDITARIA, THE HYPERKALEMIC FORM OF PERIODIC PARALYSIS].[遗传性发作性无力症（周期性麻痹的高钾血症形式）中的电解质与醛固酮代谢]
Klin Wochenschr. 1964 Jan 15;42:65-75. doi: 10.1007/BF01478655.
3
[Role of adosterone in paroxysmal paralysis].[醛固酮在阵发性麻痹中的作用]
Klin Wochenschr. 1963 Feb 15;41:178-84. doi: 10.1007/BF01479429.
4
[Metabolic changes and K42 exchange in paroxymal paralysis].
Klin Wochenschr. 1963 Jan 1;41:6-10. doi: 10.1007/BF01478610.
5
Nutritional factors in the pathogenesis of cardiac necroses. II.心脏坏死发病机制中的营养因素。II.
Z Ernahrungswiss. 1962 Oct;3:1-26. doi: 10.1007/BF02021335.
6
Unusual responses to drugs in some hereditary conditions.某些遗传性疾病对药物的异常反应。
Can Anaesth Soc J. 1961 Jan;8:43-52. doi: 10.1007/BF03015389.
7
[On the hypokalemic form of paroxysmal paralysis. On the problem hereditary of episodic adynamial].
Dtsch Z Nervenheilkd. 1961;182:92-118.
8
Potassium metabolism and gastrointestinal function; a review; a semiquantitative approach.钾代谢与胃肠功能；综述；一种半定量方法
Am J Dig Dis. 1959 Feb;4(2):145-58. doi: 10.1007/BF02231235.
9
Quinidine poisoning as a cause of generalized muscle weakness.奎尼丁中毒作为全身肌无力的一个病因。
Br Med J. 1959 Jan 31;1(5117):281-2. doi: 10.1136/bmj.1.5117.281.
10
Discussion on some clinical, genetic and biochemical aspects of metabolic disorders of the nervous system: metabolic disease of the nervous system: clinical aspects.神经系统代谢紊乱的一些临床、遗传和生化方面的探讨：神经系统代谢疾病：临床方面
Proc R Soc Med. 1958 Oct;51(10):859-63. doi: 10.1177/003591575805101014.