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The effect of triton and diphenylylethylacetic acid on cholesterol and fatty acid biosynthesis in isolated perfused liver.

作者信息

GARATTINI S, PAOLETTI P, PAOLETTI R

出版信息

Experientia. 1959 Jan 15;15(1):33-4. doi: 10.1007/BF02157091.

DOI:10.1007/BF02157091
PMID:13619684
Abstract
摘要

相似文献

1
The effect of triton and diphenylylethylacetic acid on cholesterol and fatty acid biosynthesis in isolated perfused liver.曲拉通和二苯乙基乙酸对离体灌注肝脏中胆固醇和脂肪酸生物合成的影响。
Experientia. 1959 Jan 15;15(1):33-4. doi: 10.1007/BF02157091.
2
The effect of diphenylylethylacetic acid on cholesterol and fatty acid biosynthesis.二苯乙基乙酸对胆固醇和脂肪酸生物合成的影响。
Arch Int Pharmacodyn Ther. 1958 Oct 1;117(1-2):114-22.
3
Effect of some hyper- and hypocholesteremic drugs on the cholesterol biosynthesis by liver homogenates.某些高胆固醇血症和低胆固醇血症药物对肝匀浆胆固醇生物合成的影响。
Experientia. 1961 May 15;17:222. doi: 10.1007/BF02160628.
4
Acceleration of hepatic cholesterol synthesis by triton WR-1339.曲拉通WR - 1339对肝脏胆固醇合成的促进作用。
J Exp Med. 1955 Mar 1;101(3):225-32. doi: 10.1084/jem.101.3.225.
5
Comparison of hydrophobic surfactant and cholestyramine on lipid and sterol balance in the rat.疏水性表面活性剂与消胆胺对大鼠脂质和甾醇平衡的比较
Exp Mol Pathol. 1980 Oct;33(2):223-30. doi: 10.1016/0014-4800(80)90021-0.
6
Studies on biosynthesis of cholesterol. 8. The effect of Mg2+ and Mn2+ ions and of adenosine triphosphate on enzymic activity of microsomes in sterol biosynthesis from mevalonic acid.胆固醇生物合成的研究。8. Mg2+和Mn2+离子以及三磷酸腺苷对微粒体从甲羟戊酸合成甾醇的酶活性的影响。
Biochem J. 1959 Apr;71(4):660-5. doi: 10.1042/bj0710660.
7
The pathogenesis of hyperlipemia induced by means of surface-active agents. II. Failure of exchange of cholesterol between the plasma and the liver in rabbits given triton WR 1339.表面活性剂所致高脂血症的发病机制。II. 给予曲通WR 1339的家兔血浆与肝脏之间胆固醇交换的障碍
J Exp Med. 1956 Jul 1;104(1):15-24. doi: 10.1084/jem.104.1.15.
8
[On cholesterin and fatty acid synthesis in the diabetic rat liver].[关于糖尿病大鼠肝脏中胆固醇和脂肪酸的合成]
Hoppe Seylers Z Physiol Chem. 1961 May 30;324:101-7. doi: 10.1515/bchm2.1961.324.1.101.
9
The pathogenesis of hyperlipemia induced by means of surface-active agents. I. Increased total body cholesterol in mice given triton WR 1339 parenterally.表面活性剂所致高脂血症的发病机制。I. 经肠胃外给予小鼠曲通WR 1339后体内总胆固醇增加。
J Exp Med. 1956 Jul 1;104(1):1-13. doi: 10.1084/jem.104.1.1.
10
Fatty acid inhibition of cholesterol synthesis.脂肪酸对胆固醇合成的抑制作用。
Can J Biochem Physiol. 1956 Sep;34(5):861-8.

本文引用的文献

1
Cholesterol studies. I. The cholesterol content of foods.胆固醇研究。一、食物中的胆固醇含量。
Scand J Clin Lab Invest. 1952;4(2):115-21. doi: 10.3109/00365515209060646.
2
The dominant role of the liver in plasma protein synthesis; a direct study of the isolated perfused rat liver with the aid of lysine-epsilon-C14.肝脏在血浆蛋白合成中的主导作用；借助赖氨酸-ε-C14对离体灌注大鼠肝脏进行的直接研究。
J Exp Med. 1951 Nov;94(5):431-53. doi: 10.1084/jem.94.5.431.
3
A series of new compounds inhibiting the acetylation of choline in vitro.一系列在体外抑制胆碱乙酰化作用的新化合物。
Experientia. 1958 Mar 15;14(3):89-91. doi: 10.1007/BF02159227.
4
A series of new compounds inhibiting coenzyme A and cholesterol and lipid-increasing factors.一系列抑制辅酶A以及胆固醇和脂质增加因子的新化合物。
Arch Int Pharmacodyn Ther. 1957 Feb 1;109(3-4):400-14.
5
Diphenylilethylacetic acid inhibits hypercholesteremia induced by triton.二苯乙腈乙酸抑制 Triton 诱导的高胆固醇血症。
Experientia. 1956 Sep 15;12(9):347-8. doi: 10.1007/BF02165345.
6
Acceleration of hepatic cholesterol synthesis by triton WR-1339.曲拉通WR - 1339对肝脏胆固醇合成的促进作用。
J Exp Med. 1955 Mar 1;101(3):225-32. doi: 10.1084/jem.101.3.225.
7
Biosynthesis of fatty acids by slices and cell-free suspensions of mammary gland.乳腺切片和无细胞悬浮液中脂肪酸的生物合成
Biochem J. 1954 Jan;56(1):46-54. doi: 10.1042/bj0560046.
8
The mechanism responsible for the hypercholesteremia induced by triton WR-1339.由曲拉通WR - 1339诱导的高胆固醇血症的发病机制。
J Exp Med. 1953 Jan;97(1):117-30. doi: 10.1084/jem.97.1.117.