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Myelodysplasia and leukemia after treatment of aplastic anemia with G-CSF.

作者信息

Kojima S, Tsuchida M, Matsuyama T

出版信息

N Engl J Med. 1992 May 7;326(19):1294-5. doi: 10.1056/NEJM199205073261917.

DOI:10.1056/NEJM199205073261917
PMID:1373226
Abstract
摘要

相似文献

1
Myelodysplasia and leukemia after treatment of aplastic anemia with G-CSF.再生障碍性贫血经粒细胞集落刺激因子治疗后发生的骨髓发育异常和白血病。
N Engl J Med. 1992 May 7;326(19):1294-5. doi: 10.1056/NEJM199205073261917.
2
[Myelodysplastic syndrome with monosomy 7 following combination therapy with granulocyte colony-stimulating factor, cyclosporin A and danazole in an adult patient with severe aplastic anemia].[一名重度再生障碍性贫血成年患者在接受粒细胞集落刺激因子、环孢素A和达那唑联合治疗后出现7号染色体单体的骨髓增生异常综合征]
Rinsho Ketsueki. 1997 Sep;38(9):745-51.
3
The evidence of clonal evolution with monosomy 7 in aplastic anemia following granulocyte colony-stimulating factor using the polymerase chain reaction.使用聚合酶链反应检测粒细胞集落刺激因子治疗后再生障碍性贫血伴7号染色体单体型的克隆进化证据。
Blood Cells Mol Dis. 1997 Aug;23(2):213-8. doi: 10.1006/bcmd.1997.0138.
4
Myelodysplasia and acute myeloid leukaemia in cases of aplastic anaemia and congenital neutropenia following G-CSF administration.粒细胞集落刺激因子(G-CSF)治疗后再生障碍性贫血和先天性中性粒细胞减少症患者发生的骨髓发育异常和急性髓系白血病。
Br J Haematol. 1995 Jan;89(1):188-90. doi: 10.1111/j.1365-2141.1995.tb08928.x.
5
[Advances in the research of clinical use of recombinant human granulocyte colony-stimulating factors].
Zhonghua Nei Ke Za Zhi. 1991 Nov;30(11):723-5.
6
Kostmann's disease, recombinant HuG-CSF, monosomy 7 and MDS/AML.科茨曼病、重组人粒细胞集落刺激因子、单体7以及骨髓增生异常综合征/急性髓系白血病
Br J Haematol. 1995 Sep;91(1):150-3. doi: 10.1111/j.1365-2141.1995.tb05260.x.
7
[3-line response following long-term therapy of severe aplastic anemia using glycosylated rHuG-CSF: dysfunctional thrombocytes as early indication of a myelodysplastic syndrome].
Schweiz Med Wochenschr. 1994 Jan 11;124(1-2):69-72.
8
[RAEB in T with monosomy 7 after treatment of severe aplastic anemia with long term G-CSF].[严重再生障碍性贫血经长期粒细胞集落刺激因子治疗后出现7号染色体单体的转化型难治性贫血伴原始细胞增多]
Rinsho Ketsueki. 1995 Apr;36(4):365-70.
9
Transformation of severe aplastic anemia into myelodysplastic syndrome with monosomy 7: monoclonal origin detected by HUMARA gene analysis during the aplastic anemia phase.伴有7号染色体单体的严重再生障碍性贫血转化为骨髓增生异常综合征:在再生障碍性贫血阶段通过HUMARA基因分析检测到单克隆起源
Haematologica. 2000 Jun;85(6):665-6.
10
Myelodysplasia following aplastic anaemia-paroxysmal nocturnal haemoglobinuria syndrome after treatment with immunosuppression and G-CSF: evidence for the emergence of a separate clone.再生障碍性贫血-阵发性睡眠性血红蛋白尿综合征经免疫抑制和粒细胞集落刺激因子治疗后发生的骨髓发育异常:一个独立克隆出现的证据
Br J Haematol. 1996 Sep;94(3):510-2. doi: 10.1046/j.1365-2141.1996.d01-1831.x.

引用本文的文献

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Myeloid cells promote interferon signaling-associated deterioration of the hematopoietic system.髓系细胞促进与干扰素信号相关的造血系统恶化。
Nat Commun. 2022 Dec 10;13(1):7657. doi: 10.1038/s41467-022-35318-x.
2
Mutation, drift and selection in single-driver hematologic malignancy: Example of secondary myelodysplastic syndrome following treatment of inherited neutropenia.单驱动血液系统恶性肿瘤中的突变、漂移和选择:遗传性中性粒细胞减少症治疗后发生的继发性骨髓增生异常综合征实例。
PLoS Comput Biol. 2019 Jan 7;15(1):e1006664. doi: 10.1371/journal.pcbi.1006664. eCollection 2019 Jan.
3
Effects of granulocyte-colony-stimulating factor on Monosomy 7 aneuploidy in healthy hematopoietic stem cell and granulocyte donors.
粒细胞集落刺激因子对健康造血干细胞和粒细胞供者单体 7 非整倍体的影响。
Transfusion. 2012 Mar;52(3):537-41. doi: 10.1111/j.1537-2995.2011.03313.x. Epub 2011 Aug 29.
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Two cases of acute myeloblastic leukemia evolving from aplastic anemia.两例由再生障碍性贫血演变而来的急性髓系白血病病例。
Int J Hematol. 2003 Jun;77(5):471-5. doi: 10.1007/BF02986615.
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Prolonged bone marrow failure with monosomy 7 after engraftment failure following bone marrow transplantation.骨髓移植后植入失败后出现伴有7号染色体单体的长期骨髓衰竭。
Int J Hematol. 2001 Feb;73(2):258-61. doi: 10.1007/BF02981947.
6
Clinical toxicity of cytokines used as haemopoietic growth factors.用作造血生长因子的细胞因子的临床毒性。
Drug Saf. 1995 Dec;13(6):371-406. doi: 10.2165/00002018-199513060-00006.
7
Transformation of severe aplastic anemia into acute myeloblastic leukemia with monosomy 7.严重再生障碍性贫血转化为伴有7号染色体单体的急性髓细胞白血病。
Ann Hematol. 1996 May;72(5):337-9. doi: 10.1007/s002770050183.