Acute toxic liver injury. nicotinamide-adenine dinucleotide-pyrophosphorylase activity of nuclei isolated from rat liver in heliotrine and in dimethylnitrosamine poisoning.
作者信息
CHRISTIE G S, BAILIE M J, LE PAGE R N
出版信息
Biochem J. 1962 Aug;84(2):364-8. doi: 10.1042/bj0840364.
Abstract
摘要
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Acute toxic liver injury. nicotinamide-adenine dinucleotide-pyrophosphorylase activity of nuclei isolated from rat liver in heliotrine and in dimethylnitrosamine poisoning.
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本文引用的文献
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Liver damage in acute heliotrine poisoning. 1. The intracellular distribution of pyridine nucleotides.
Biochem J. 1962 Jul;84(1):25-38. doi: 10.1042/bj0840025.
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Diphosphopyridine nucleotide pyrophosphorylase in the nuclei isolated from poisoned and regenerating rat liver.
Biochem J. 1961 Sep;80(3):481-7. doi: 10.1042/bj0800481.
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The desoxyribose nucleic acid content of animal nuclei.
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Liver damage in acute heliotrine poisioning. 2. The effect of nicotinamide on pyridine nucleotide concentrations.
Biochem J. 1962 Jul;84(1):202-12. doi: 10.1042/bj0840202.
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The acute toxic action of dimethylnitrosamine on liver cells.
Biochem J. 1959 Jul;72(3):473-9. doi: 10.1042/bj0720473.
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Pyridine nucleotide metabolism in heliotrine poisoning.
Nature. 1961 Feb 18;189:593-4. doi: 10.1038/189593b0.
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Enlargement of liver cell nuclei: effect of dimethylnitrosamine on size and desoxyribosenucleic acid content.
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Biosynthesis of diphosphopyridine nucleotide. II. Enzymatic aspects.
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Biosynthesis of diphosphopyridine nucleotide. I. Identification of intermediates.
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Enzymic synthesis of coenzyme I in relation to chemical control of cell growth.
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