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Pathogenesis of the coagulation defect developing during pathological plasma proteolytic ("fibrinolytic") states. I. The significance of fibrinogen proteolysis and circulating fibrinogen breakdown products.

作者信息

FLETCHER A P, ALKJAERSIG N, SHERRY S

出版信息

J Clin Invest. 1962 Apr;41(4):896-916. doi: 10.1172/JCI104546.

DOI:10.1172/JCI104546
PMID:13893560
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC290991/
Abstract
摘要

相似文献

1
Pathogenesis of the coagulation defect developing during pathological plasma proteolytic ("fibrinolytic") states. I. The significance of fibrinogen proteolysis and circulating fibrinogen breakdown products.病理性血浆蛋白水解（“纤维蛋白溶解”）状态下发生的凝血缺陷的发病机制。I. 纤维蛋白原蛋白水解和循环纤维蛋白原降解产物的意义。
J Clin Invest. 1962 Apr;41(4):896-916. doi: 10.1172/JCI104546.
2
Pathogenesis of the coagulation defect developing during pathological plasma proteolytic ("fibrinolytic") states. II. The significance, mechanism and consequences of defective fibrin polymerization.病理性血浆蛋白水解（“纤维蛋白溶解”）状态下凝血缺陷的发病机制。II. 纤维蛋白聚合缺陷的意义、机制及后果。
J Clin Invest. 1962 Apr;41(4):917-34. doi: 10.1172/JCI104547.
3
Pathogenesis of the coagulation defect developing during pathological plasma proteolytic ("fibrinolytic") states. III. Demonstration of abnormal clot structure by electron microscopy.病理性血浆蛋白水解（“纤维蛋白溶解”）状态下发生的凝血缺陷的发病机制。III. 用电镜显示异常凝块结构
J Clin Invest. 1962 Apr;41(4):935-48. doi: 10.1172/JCI104548.
4
Fibrinogen and fibrin degradation products in hemolytic transfusion reactions.溶血性输血反应中的纤维蛋白原和纤维蛋白降解产物。
Transfusion. 1970 Nov-Dec;10(6):317-21. doi: 10.1111/j.1537-2995.1970.tb00751.x.
5
[Fibrinolysis and fibrinolytic hemorrhagic diatheses].
J Med Lyon. 1952 Oct 5;33(786):869-81.
6
Fibrinogen Marburg a new genetic variant of fibrinogen.纤维蛋白原马尔堡——一种新的纤维蛋白原基因变体。
Blut. 1977 Feb;34(2):107-18. doi: 10.1007/BF00999857.
7
CURRENT STATUS OF THROMBOLYTIC THERAPY.
Geriatrics. 1964 Sep;19:628-43.
8
[Fibrinolysis as a cause of hemorrhagic diathesis].
Ugeskr Laeger. 1956 Apr 26;118(15):403-8.
9
[Hypodysfibrinogenemia: fibrinogen giessen II (author's transl)].
Klin Wochenschr. 1975 Aug 15;53(16):781-2. doi: 10.1007/BF01614861.
10
[BLOOD COAGULATION DISORDERS DURING LABOR WITH SPECIAL REFERENCE TO FIBRINOLYSIS].
Geburtshilfe Frauenheilkd. 1965 Jan;25:46-55.

引用本文的文献

1
Thrombolytic Therapy.溶栓治疗
Br Med J. 1963 Jun 8;1(5344):1492-4.
2
TO-DAY'S DRUGS. FIBRINOLYTIC ENZYMES AND FIBRINOLYTIC INHIBITORS.今日药物。纤维蛋白溶解酶与纤维蛋白溶解抑制剂。
Br Med J. 1965 Mar 13;1(5436):704-6.
3
HAEMORRHAGE AND FIBRINOLYSIS.出血与纤维蛋白溶解
Proc R Soc Med. 1964 Jul;57(7):603-6. doi: 10.1177/003591576405700733.
4
ABNORMAL PLASMINOGEN-PLASMIN SYSTEM ACTIVITY (FIBRINOLYSIS) IN PATIENTS WITH HEPATIC CIRRHOSIS: ITS CAUSE AND CONSEQUENCES.肝硬化患者纤溶酶原-纤溶酶系统活性异常（纤维蛋白溶解）：其原因及后果
J Clin Invest. 1964 Apr;43(4):681-95. doi: 10.1172/JCI104953.
5
CURRENT APPRAISAL OF THROMBOLYTIC THERAPY. A CRITICAL REVIEW.溶栓治疗的当前评估。一项批判性综述。
Calif Med. 1963 Nov;99(5):301-7.
6
CIRCULATING FIBRINOLYTIC ACTIVATOR AND HEMORRHAGIC DIATHESIS.循环纤维蛋白溶解激活剂与出血素质
Ann Surg. 1963 Jul;158(1):117-25. doi: 10.1097/00000658-196307000-00021.
7
[Thromboelastographic studies on the blood clot structure in the Sanarelli-Shwartzman phenomenon].[关于桑纳雷利-施瓦茨曼现象中血凝块结构的血栓弹力图研究]
Klin Wochenschr. 1963 Mar 1;41:216-9. doi: 10.1007/BF01487695.
8
Thrombolytic therapy.溶栓治疗。
Proc R Soc Med. 1963 May;56(5):414-8. doi: 10.1177/003591576305600536.
9
Pathogenesis of the coagulation defect developing during pathological plasma proteolytic ("fibrinolytic") states. III. Demonstration of abnormal clot structure by electron microscopy.病理性血浆蛋白水解（“纤维蛋白溶解”）状态下发生的凝血缺陷的发病机制。III. 用电镜显示异常凝块结构
J Clin Invest. 1962 Apr;41(4):935-48. doi: 10.1172/JCI104548.
10
Pathogenesis of the coagulation defect developing during pathological plasma proteolytic ("fibrinolytic") states. II. The significance, mechanism and consequences of defective fibrin polymerization.病理性血浆蛋白水解（“纤维蛋白溶解”）状态下凝血缺陷的发病机制。II. 纤维蛋白聚合缺陷的意义、机制及后果。
J Clin Invest. 1962 Apr;41(4):917-34. doi: 10.1172/JCI104547.

本文引用的文献

1
Prothrombin and Fibrinolysin.凝血酶原与纤维蛋白溶酶。
Science. 1946 Nov 15;104(2707):461-2. doi: 10.1126/science.104.2707.461.
2
Thrombin formation; effects of lysin (fibrinolysin, plasmin) on prothrombin, Ac-globulin and tissue thromboplastin.凝血酶的形成；溶素（纤维蛋白溶酶、纤溶酶）对凝血酶原、Ac球蛋白和组织凝血活酶的影响。
J Clin Invest. 1949 Nov;28(6 Pt 2):1507-10. doi: 10.1172/JCI102216.
3
The protease and antiprotease of plasmas of patients with cancer and other diseases.癌症及其他疾病患者血浆中的蛋白酶和抗蛋白酶。
Cancer Res. 1949 Nov;9(11):665-8.
4
[Fatal hemorrhagic syndromes with total incoagulability and with fibrinolysis; in pulmonary exeresis].
Rev Hematol. 1952;7(1):30-47.
5
Studies on a proteolytic enzyme in human plasma. VII. A fatal hemorrhagic state associated with excessive plasma proteolytic activity in a patient undergoing surgery for carcinoma of the head of the pancreas.人血浆中一种蛋白水解酶的研究。VII. 一名接受胰头癌手术患者出现与血浆蛋白水解活性过高相关的致命出血状态。
J Clin Invest. 1952 May;31(5):521-8. doi: 10.1172/JCI102636.
6
Effects of ions and neutral molecules on fibrin clotting.离子和中性分子对纤维蛋白凝血的影响。
J Biol Chem. 1951 Aug;191(2):735-56.
7
Studies on afibrinogenemia. II. The defibrinating effect on dog blood of intravenous injection of thromboplastic material.
Bull Johns Hopkins Hosp. 1951 May;88(5):414-24.
8
A new method for the determination of fibrinogen in small samples of plasma.一种测定少量血浆样本中纤维蛋白原的新方法。
J Lab Clin Med. 1951 Feb;37(2):316-20.
9
Chemical, clinical and immunological studies on the products of human plasma fractionation XL. Quantitative separation and determination of the protein components in small amounts of normal human plasma.人血浆成分制品的化学、临床及免疫学研究XL。少量正常人血浆中蛋白质成分的定量分离与测定。
J Clin Invest. 1951 Jan;30(1):99-111. doi: 10.1172/JCI102423.
10
Etiology of fibrinopenia: fibrination defibrination.纤维蛋白减少症的病因：纤维蛋白形成与纤维蛋白溶解。
Ann N Y Acad Sci. 1959 Jan 9;75:634-75. doi: 10.1111/j.1749-6632.1959.tb44581.x.