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Hemodynamic alterations due to Salmonella typhosa endotoxin with special reference to the coronary vascular bed.

作者信息

FROHLICH E D, SCOTT J B, DOOLEY E S

出版信息

J Clin Invest. 1962 Jan;41(1):147-52. doi: 10.1172/JCI104457.

DOI:10.1172/JCI104457
PMID:13895460
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC289204/
Abstract
摘要

相似文献

1
Hemodynamic alterations due to Salmonella typhosa endotoxin with special reference to the coronary vascular bed.伤寒沙门氏菌内毒素引起的血流动力学改变，特别涉及冠状血管床。
J Clin Invest. 1962 Jan;41(1):147-52. doi: 10.1172/JCI104457.
2
Hemodynamics of the stomach. III. Effects of Salmonella typhosa endotoxin on the resistance to blood flow.胃的血液动力学。III. 伤寒沙门氏菌内毒素对血流阻力的影响。
Rep US Army Med Res Lab. 1961 Oct 19:9p. doi: 10.21236/ad0265967.
3
Species differences in effect of gram-negative endotoxin on circulation.革兰氏阴性内毒素对循环系统作用的种属差异。
Am J Physiol. 1961 Jun;200:1197-202. doi: 10.1152/ajplegacy.1961.200.6.1197.
4
[Vascular permeability and endothelial adhesiveness of the dermis of rabbits treated with S. typhi endotoxin and basic proteins].[伤寒沙门氏菌内毒素和碱性蛋白处理的家兔真皮血管通透性和内皮粘附性]
Minerva Dermatol. 1961 Jul;36:219-24.
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Hemodynamic alterations due to Salmonella typhosa endotoxin with special reference to the coronary vascular bed.伤寒沙门氏菌内毒素引起的血流动力学改变，特别涉及冠状动脉床。
Rep US Army Med Res Lab. 1961 May 29;481:1-11. doi: 10.21236/ad0263252.
6
Inactivation of endotoxin by a humoral component. VI. Two separate systems required for viable and killed Salmonella typhosa.体液成分对内毒素的灭活作用。VI. 活的和灭活的伤寒沙门氏菌所需的两个独立系统。
J Clin Invest. 1960 Feb;39(2):352-63. doi: 10.1172/JCI104046.
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Intestinal tissue blood flow in shock due to endotoxin.内毒素所致休克时的肠组织血流
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[On the significance of endotoxin in experimental typhoid. I. Extraction of endotoxin from mutants of Salmonella enteritidis and the effect of the endotoxin on infection in mice caused by those strains].[关于内毒素在实验性伤寒中的意义。I. 肠炎沙门氏菌突变株内毒素的提取及其对由这些菌株引起的小鼠感染的影响]
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[Effect of a sublethal dose of endotoxin on the renal circulation of the rabbit].[亚致死剂量内毒素对家兔肾循环的影响]
C R Seances Soc Biol Fil. 1961;155:2078-83.
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[Cardiotropic electivity in endotoxin-noradrenalin synergism].[内毒素 - 去甲肾上腺素协同作用中的心脏选择性]
Pathologica. 1960 Sep-Oct;52:369-77.

引用本文的文献

1
VASOACTIVE MEDIATORS AS THE "TRIGGER MECHANISM" OF ENDOTOXIN SHOCK.血管活性介质作为内毒素休克的“触发机制” 。
J Clin Invest. 1964 May;43(5):1000-13. doi: 10.1172/JCI104963.
2
Effects of endotoxin on the hemodynamics of the stomach.内毒素对胃血流动力学的影响。
J Clin Invest. 1963 Mar;42(3):391-5. doi: 10.1172/JCI104725.

本文引用的文献

1
Role of the non-Newtonian behavior of blood in hemodynamics.血液的非牛顿行为在血流动力学中的作用。
Am J Physiol. 1959 Nov;197:943-50. doi: 10.1152/ajplegacy.1959.197.5.943.
2
Mechanisms of the hemodynamic effects of endotoxin.内毒素血流动力学效应的机制。
Physiol Rev. 1960 Apr;40:245-79. doi: 10.1152/physrev.1960.40.2.245.
3
Peripheral resistance changes and blood pooling after endotoxin in eviscerated dogs.去内脏犬内毒素注射后外周阻力变化及血液淤积
Am J Physiol. 1958 Dec;195(3):631-4. doi: 10.1152/ajplegacy.1958.195.3.631.
4
The intestinal factor in irreversible endotoxin shock.不可逆性内毒素休克中的肠道因素。
Ann Surg. 1958 Oct;148(4):513-24; discussion 524-5. doi: 10.1097/00000658-195810000-00002.
5
Influence of perfusate characteristics on pulmonary vascular response to endotoxin.灌注液特性对肺血管内毒素反应的影响。
Am J Physiol. 1957 Nov;191(2):293-5. doi: 10.1152/ajplegacy.1957.191.2.293.
6
Alterations in kidney weight produced by Escherichia coli endotoxin.大肠杆菌内毒素引起的肾脏重量变化。
Am J Physiol. 1957 May;189(2):329-30. doi: 10.1152/ajplegacy.1957.189.2.329.
7
Studies on the circulatory changes in the dog produced by endotoxin from gram-negative microorganisms.革兰氏阴性微生物内毒素所致犬循环系统变化的研究。
J Clin Invest. 1956 Nov;35(11):1191-8. doi: 10.1172/JCI103373.
8
Canine intestinal and liver weight changes induced by E. coli endotoxin.大肠杆菌内毒素诱导的犬肠道和肝脏重量变化。
Proc Soc Exp Biol Med. 1956 Jul;92(3):602-5. doi: 10.3181/00379727-92-22557.
9
Hypotension (shock) in dogs produced by Escherichia coli endotoxin.大肠杆菌内毒素所致犬的低血压（休克）
Circ Res. 1956 Sep;4(5):546-56. doi: 10.1161/01.res.4.5.546.
10
The physiological disturbances produced by endotoxins.内毒素产生的生理紊乱。
Annu Rev Physiol. 1954;16:467-90. doi: 10.1146/annurev.ph.16.030154.002343.