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Endotoxin shock. A manifestation of intravascular coagulation.

作者信息

HARDAWAY R M, HUSNI E A, GEEVER E F, BURNS J W

出版信息

Ann Surg. 1961 Nov;154(5):791-802. doi: 10.1097/00000658-196111000-00003.

DOI:10.1097/00000658-196111000-00003
PMID:13904590
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1465952/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/52459c116dda/annsurg00859-0063-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/5f4b634c05dd/annsurg00859-0056-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/05b18ff08952/annsurg00859-0056-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/1fc648ea92d5/annsurg00859-0057-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/294dbfe214e5/annsurg00859-0057-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/45d66ce17670/annsurg00859-0057-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/47a79d91e090/annsurg00859-0058-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/63e2198ad16f/annsurg00859-0058-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/100c569cc43b/annsurg00859-0058-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/dbf46f62fb9f/annsurg00859-0059-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/413be6aa8475/annsurg00859-0059-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/c5b54235419a/annsurg00859-0059-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/e40d4a5b7201/annsurg00859-0060-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/1c852885c49a/annsurg00859-0061-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/25b924b28ce4/annsurg00859-0061-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/eb0ac6ed7281/annsurg00859-0062-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/dfbfd4e26b5a/annsurg00859-0063-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/52459c116dda/annsurg00859-0063-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/5f4b634c05dd/annsurg00859-0056-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/05b18ff08952/annsurg00859-0056-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/1fc648ea92d5/annsurg00859-0057-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/294dbfe214e5/annsurg00859-0057-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/45d66ce17670/annsurg00859-0057-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/47a79d91e090/annsurg00859-0058-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/63e2198ad16f/annsurg00859-0058-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/100c569cc43b/annsurg00859-0058-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/dbf46f62fb9f/annsurg00859-0059-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/413be6aa8475/annsurg00859-0059-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/c5b54235419a/annsurg00859-0059-c.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/e40d4a5b7201/annsurg00859-0060-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/1c852885c49a/annsurg00859-0061-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/25b924b28ce4/annsurg00859-0061-b.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/eb0ac6ed7281/annsurg00859-0062-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/dfbfd4e26b5a/annsurg00859-0063-a.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7f02/1465952/52459c116dda/annsurg00859-0063-b.jpg

相似文献

1
Endotoxin shock. A manifestation of intravascular coagulation.内毒素休克。一种血管内凝血的表现。
Ann Surg. 1961 Nov;154(5):791-802. doi: 10.1097/00000658-196111000-00003.
2
Experimental endotoxin shock. Circulatory changes with emphasis upon cardiac function.实验性内毒素休克。循环系统变化，重点关注心脏功能。
Am J Surg. 1962 Jun;103:702-8. doi: 10.1016/0002-9610(62)90249-0.
3
Role of hypotension in altering blood coagulability after endotoxin.低血压在内毒素后改变血液凝固性中的作用。
Am J Physiol. 1962 Mar;202:519-22. doi: 10.1152/ajplegacy.1962.202.3.519.
4
Mechanism of histamine release in endotoxin shock.内毒素休克中组胺释放的机制。
Am J Physiol. 1961 May;200:987-9. doi: 10.1152/ajplegacy.1961.200.5.987.
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Effect of endotoxin on the clotting mechanism. II. On the variation in response in different species of animals.内毒素对凝血机制的影响。II. 不同动物物种反应的变化
Ann Surg. 1961 Mar;153(3):453-8. doi: 10.1097/00000658-196103000-00018.
6
Role of histamine in endotoxin shock.组胺在内毒素休克中的作用。
Proc Soc Exp Biol Med. 1960 Jul;104:379-81. doi: 10.3181/00379727-104-25843.
7
Adrenal catecholamines in E. coli endotoxin shock.大肠杆菌内毒素休克中的肾上腺儿茶酚胺
J Appl Physiol. 1961 Mar;16:348-50. doi: 10.1152/jappl.1961.16.2.348.
8
Effects of aldosterone in therapy of endotoxin shock.醛固酮在内毒素休克治疗中的作用。
Surg Forum. 1961;12:92-4.
9
Role of the intestine in endotoxin shock.肠道在内毒素休克中的作用。
Proc Soc Exp Biol Med. 1962 Jan;109:200-2. doi: 10.3181/00379727-109-27151.
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Carotid sinus baroceptor modifications associated with endotoxin shock.与内毒素休克相关的颈动脉窦压力感受器改变
Am J Physiol. 1962 May;202:971-7. doi: 10.1152/ajplegacy.1962.202.5.971.

引用本文的文献

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STUDIES ON THE FIBRINOGEN REPLACEMENT RATE IN DOGS.犬纤维蛋白原替代率的研究。
Ann Surg. 1964 Nov;160(5):835-8. doi: 10.1097/00000658-196411000-00009.
2
VASOACTIVE MEDIATORS AS THE "TRIGGER MECHANISM" OF ENDOTOXIN SHOCK.血管活性介质作为内毒素休克的“触发机制” 。
J Clin Invest. 1964 May;43(5):1000-13. doi: 10.1172/JCI104963.
3
CAUSES OF DEATH IN MESENTERIC ARTERIAL OCCLUSION. II. OBSERVATIONS ON REVASCULARIZATION OF THE ISCHEMIC BOWEL.肠系膜动脉闭塞的死亡原因。II. 缺血肠管血运重建的观察

本文引用的文献

1
Some aspects of the fibrinogen-fibrin transition.纤维蛋白原-纤维蛋白转变的某些方面。
Nature. 1960 Aug 27;187:758-61. doi: 10.1038/187758a0.
2
Precipitation of human fibrinogen with heparin.用肝素沉淀人纤维蛋白原。
Scalpel (Brux). 1960;12:56-65.
3
Alterations in blood coagulation mechanism after intra-aortic injection of thrombin.主动脉内注射凝血酶后血液凝固机制的改变。
Ann Surg. 1963 Dec;158(6):960-70. doi: 10.1097/00000658-196312000-00006.
4
THE ROLE OF MICRO-EMBOLISM IN THE PRODUCTION OF IRREVERSIBLE SHOCK.微栓塞在不可逆性休克产生中的作用
Ann Surg. 1963 Oct;158(4):685-97. doi: 10.1097/00000658-196310000-00015.
5
The role of the central nervous system in septic shock. II. Hemodynamic, respiratory and metabolic effects of intracisternal or intraventricular endotoxin.中枢神经系统在脓毒性休克中的作用。II. 脑池内或脑室内注入内毒素的血流动力学、呼吸及代谢效应
Ann Surg. 1968 Feb;167(2):158-67. doi: 10.1097/00000658-196802000-00002.
6
Molecular aspects of endotoxic reactions.内毒素反应的分子层面
Bacteriol Rev. 1969 Mar;33(1):72-98. doi: 10.1128/br.33.1.72-98.1969.
7
Sepsis and gastrointestinal bleeding.脓毒症与胃肠道出血。
Ann Surg. 1972 May;175(5):759-70. doi: 10.1097/00000658-197205000-00016.
8
Significane of intravascular coagulation in canine endotoxin shock.犬内毒素休克中血管内凝血的意义
Infect Immun. 1975 May;11(5):1010-3. doi: 10.1128/iai.11.5.1010-1013.1975.
9
Experimental gram-negative bacterial sepsis: prevention of mortality not preventable by antibiotics alone.实验性革兰氏阴性菌败血症：仅靠抗生素无法预防的死亡率的预防。
Infect Immun. 1979 Aug;25(2):538-57. doi: 10.1128/iai.25.2.538-557.1979.
10
Endotoxin toxicity in rats with 6-sulfanilamidoindazole arthritis.患有6-磺胺氨基吲唑关节炎的大鼠的内毒素毒性
Infect Immun. 1979 Jul;25(1):337-44. doi: 10.1128/iai.25.1.337-344.1979.
Arch Surg. 1960 Dec;81:983-91. doi: 10.1001/archsurg.1960.01300060129025.
4
Intravascular thrombi and the intestinal factor of irreversible shock.血管内血栓与不可逆休克的肠道因素。
Ann Surg. 1959 Aug;150(2):261-5. doi: 10.1097/00000658-195908000-00008.
5
Disseminated intravascular coagulation: a cause of shock.弥散性血管内凝血：休克的一个原因。
Ann Surg. 1959 Apr;149(4):462-70. doi: 10.1097/00000658-195904000-00002.
6
The intestinal factor in irreversible endotoxin shock.不可逆性内毒素休克中的肠道因素。
Ann Surg. 1958 Oct;148(4):513-24; discussion 524-5. doi: 10.1097/00000658-195810000-00002.
7
The detection of bacterial endotoxins in the plasma of dogs with experimental peritonitis.实验性腹膜炎犬血浆中细菌内毒素的检测
Ann Surg. 1956 Jul;144(1):51-6. doi: 10.1097/00000658-195607000-00008.
8
Pathologic study of intravascular coagulation following incompatible blood transfusion in dogs. I. Intravenous injection of incompatible blood.犬输入不相合血液后血管内凝血的病理学研究。I. 静脉注射不相合血液。
Am J Surg. 1956 Jan;91(1):24-31. doi: 10.1016/0002-9610(56)90129-5.
9
Alterations in blood coagulation mechanism after incompatible blood transfusion; clinical and experimental observations.异型输血后血液凝固机制的改变；临床与实验观察
Am J Surg. 1955 Mar;89(3):583-92. doi: 10.1016/0002-9610(55)90105-7.