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1
Prevention of "irreversible" hemorrhagic shock with fibrinolysin.
Ann Surg. 1963 Jan;157(1):39-47. doi: 10.1097/00000658-196301000-00004.
2
Mechanism of action of fibrinolysin in the prevention of irreversible hemorrhagic shock.
Ann Surg. 1963 Feb;157(2):305-9. doi: 10.1097/00000658-196302000-00020.
3
CLOTTING TIME OF HEPARINIZED BLOOD. INFLUENCE OF ACIDOSIS.
Arch Surg. 1964 Oct;89:701-5. doi: 10.1001/archsurg.1964.01320040117020.
4
THE ROLE OF MICRO-EMBOLISM IN THE PRODUCTION OF IRREVERSIBLE SHOCK.
Ann Surg. 1963 Oct;158(4):685-97. doi: 10.1097/00000658-196310000-00015.
5
Influence of fibrinolysin on shock.
JAMA. 1963 Feb 16;183:597-9. doi: 10.1001/jama.1963.63700070034020a.
6
EFFECT OF LOW MOLECULAR WEIGHT DEXTRANS AND FIBRINOLYSINS ON HEMORRHAGIC SHOCK.
Surg Forum. 1964;15:32-4.
7
SIMPLE METHOD FOR ESTABLISHING HEMORRHAGIC SHOCK IN DOGS.
Arch Surg. 1965 Jun;90:893-4. doi: 10.1001/archsurg.1965.01320120095008.
8
TREATMENT OF EXPERIMENTAL HEMORRHAGIC SHOCK.
BMQ. 1964 Jun;15:60-3.
9
Experimental and clinical experience with the therapeutic use of fibrinolysis in hemorrhagic shock.
J Cardiovasc Surg (Torino). 1967 Mar-Apr;8(2):167-70.
10
Mechanism of traumatic shock.
Surg Gynecol Obstet. 1980 Jul;151(1):65-9.
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Does Tranexamic Acid Improve Clot Strength in Severely Injured Patients Who Have Elevated Fibrin Degradation Products and Low Fibrinolytic Activity, Measured by Thrombelastography?
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Targeting resuscitation to normalization of coagulating status: Hyper and hypocoagulability after severe injury are both associated with increased mortality.
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Tranexamic acid is associated with increased mortality in patients with physiological fibrinolysis.
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The identity and origin of the anticoagulant of anaphylactic shock in the dog.
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The control of dicumarol therapy and the quantitative determination of prothrombin and proconvertin.
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[The effect of fibrinolysis on the courses of hemorrhagic shock].
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Studies on the role of intravascular coagulation in irreversible hemorrhagic shock.
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Methods for the evaluation of human fibrinolysis; studies with two combined technics.
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Intravascular clotting and afibrinogenemia, the presumptive lethal factors in the syndrome of amniotic fluid embolism.
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