Shechtman F G, Lin P T, Pincus R L
Department of Otolaryngology--Head and Neck Surgery, New York Medical College Affiliated Hospitals Program, NY 10003.
Int J Pediatr Otorhinolaryngol. 1992 Jul;24(1):83-9. doi: 10.1016/0165-5876(92)90069-2.
Adenotonsillar hypertrophy has been documented to cause chronic upper airway obstruction resulting in cardiopulmonary sequelae in children. It has been less recognized that long-term adenotonsillar hypertrophy may additionally cause acute, life-threatening airway obstruction. A review of 5000 adenotonsillectomies performed at 3 New York Medical College affiliated hospitals from 1982 to 1989 showed 6 pediatric patients with progressive upper airway obstruction severe enough to necessitate intubation in the emergency room or operating room, and subsequent urgent adenotonsillectomy after cardiorespiratory stabilization. Patients were monitored in the ICU with pre- and postoperative blood gases. Observations of cyanosis, cor pulmonale, and use of accessory respiratory muscles were carefully recorded. This study illustrates that life-threatening upper airway obstruction may be due to chronic adenotonsillar enlargement and require treatment by urgent adenotonsillectomy.
腺样体扁桃体肥大已被证实会导致儿童慢性上呼吸道阻塞,进而引发心肺后遗症。长期腺样体扁桃体肥大还可能导致急性、危及生命的气道阻塞,这一点却较少被认识到。回顾1982年至1989年在纽约医学院附属的3家医院进行的5000例腺样体扁桃体切除术,发现有6例儿科患者出现进行性上呼吸道阻塞,严重到需要在急诊室或手术室进行插管,随后在心肺功能稳定后紧急进行腺样体扁桃体切除术。患者在重症监护病房接受监测,术前和术后均进行血气分析。仔细记录了发绀、肺心病和辅助呼吸肌使用情况的观察结果。这项研究表明,危及生命的上呼吸道阻塞可能是由于慢性腺样体扁桃体肿大引起的,需要通过紧急腺样体扁桃体切除术进行治疗。