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A REVERSIBLE SALT-WASTING SYNDROME OF THE NEWBORN AND INFANT: POSSIBLE INFANTILE HYPOALDOSTERONISM.新生儿及婴儿的一种可逆性失盐综合征：可能为婴儿型醛固酮减少症。
Arch Dis Child. 1963 Aug;38(200):313-25. doi: 10.1136/adc.38.200.313.
2
[CORTICOSTEROID CLEARANCE IN ADRENAL CORTEX DISEASES. INVESTIGATIONS WITH ACTH, CORTISONE, ALDOSTERONE AND HYDROCHLOROTHIAZIDE].
Folia Endocrinol. 1964 Oct;17:568-86.
3
TREATMENT OF ADRENAL INSUFFICIENCY.
GP. 1964 Mar;29:135-40.
4
CONGENITAL ADRENAL HYPERPLASIA.先天性肾上腺皮质增生症
Arch Dis Child. 1964 Aug;39(206):324-33. doi: 10.1136/adc.39.206.324.
5
A NEW HEREDITARY DEFECT IN THE BIOSYNTHESIS OF ALDOSTERONE: URINARY C21-CORTICOSTEROID PATTERN IN THREE RELATED PATIENTS WITH A SALT-LOSING SYNDROME, SUGGESTING AN 18-OXIDATION DEFECT.
Acta Endocrinol (Copenh). 1964 Dec;47:589-612. doi: 10.1530/acta.0.0470589.
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[THE TREATMENT OF GRADUAL ADRENAL INSUFFICIENCY].[慢性肾上腺皮质功能减退症的治疗]
Cah Coll Med Hop Paris. 1963 Dec 15;4:849-59.
7
[EXCRETION OF ELECTROLYTES IN ADRENOCORTICAL INSUFFICIENCY DURING THE 4-HOUR ACTH TEST].
Pol Tyg Lek. 1964 Mar 2;19:342-6.
8
ADRENOCORTICAL CONTROL OF RENAL SODIUM AND POTASSIUM EXCRETION IN THE NEWBORN PERIOD.新生儿期肾上腺皮质对肾脏钠和钾排泄的控制
Maandschr Kindergeneeskd. 1964 Jun;32:287-315.
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[CLINICO-MORPHOLOGICAL STUDY OF ADRENAL CHANGES IN RHEUMATIC CHILDREN].[风湿性疾病患儿肾上腺变化的临床形态学研究]
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[ADDISON'S DISEASE WITH CEREBRAL SCLEROSIS IN CHILDHOOD. A HEREDITARY SYNDROME TRANSMITTED THROUGH CHROMOSOME X?].
Helv Paediatr Acta. 1963 Dec;18:480-501.

引用本文的文献

1
The adrenal cortex in childhood. 2. Pathological aspects.儿童期的肾上腺皮质。2. 病理学方面。
Arch Dis Child. 1966 Apr;41(216):113-36 contd. doi: 10.1136/adc.41.216.113.
2
Urosepsis in infants with vesicoureteral reflux masquerading as the salt-losing type of congenital adrenal hyperplasia.
Pediatr Radiol. 1989;19(8):548-50. doi: 10.1007/BF02389571.
3
Hypoaldosteronism in three sibs due to 18-dehydrogenase deficiency.三例同胞因18-脱氢酶缺乏导致醛固酮减少症。
Arch Dis Child. 1976 Aug;51(8):576-83. doi: 10.1136/adc.51.8.576.
4
Salt-losing syndrome in 2 infants with defective 18-dehydrogenation in aldosterone biosynthesis.2例醛固酮生物合成中18-脱氢缺陷婴儿的失盐综合征
Arch Dis Child. 1977 Jul;52(7):580-6. doi: 10.1136/adc.52.7.580.

本文引用的文献

1
Adrenal cortical function in newly-born infants.
J Clin Endocrinol Metab. 1950 Aug;10(8):845-57. doi: 10.1210/jcem-10-8-845.
2
Investigations on the chemical determination of corticoids in urine.尿液中皮质类固醇的化学测定研究。
Acta Endocrinol (Copenh). 1950;4(3):205-28. doi: 10.1530/acta.0.0040205.
3
[ACTH test of the infantile adrenal cortex lasting 48 hours].[婴儿肾上腺皮质持续48小时的促肾上腺皮质激素试验]
Helv Paediatr Acta. 1952 Mar;7(1):60-9.
4
The diuretic response to ingested water in Addison's disease and panhypopituitarism and the effect of cortisone thereon.艾迪生病和全垂体功能减退症对摄入水分的利尿反应以及可的松对此的影响。
Clin Sci. 1952 May;11(2):113-28.
5
[Role of the adrenal cortex in the regulation of the excretion of salt and water, studied in the rat with the aid of radiosodium].[肾上腺皮质在盐和水排泄调节中的作用，借助放射性钠在大鼠身上进行的研究]
Ann Endocrinol (Paris). 1951;12(5):704-15.
6
Effect of oral cortisone on water diuresis in Addison's disease and hypopituitarism.口服可的松对艾迪生病和垂体功能减退症水利尿作用的影响。
Lancet. 1951 Oct 13;2(6685):664-6. doi: 10.1016/s0140-6736(51)92932-7.
7
Adrenocortical control of sodium and potassium excretion in the newborn period.新生儿期肾上腺皮质对钠和钾排泄的控制
J Clin Invest. 1951 Mar;30(3):318-24. doi: 10.1172/JCI102447.
8
A salt-losing syndrome in infancy. Pseudo-hypoadrenocorticalism.婴儿期失盐综合征。假性肾上腺皮质功能减退症。
Arch Dis Child. 1962 Oct;37(195):548-56. doi: 10.1136/adc.37.195.548.
9
[Saline diabetes caused by congenital insensitivity of the tubule to aldosterone: "pseudo-hypoadrenocorticism"].[先天性肾小管对醛固酮不敏感所致的盐性糖尿病：“假性肾上腺皮质功能减退症”]
Rev Fr Etud Clin Biol. 1960 Jun-Jul;5:558-65.
10
Pseudohypo-adrenalocorticism; renal sodium loss, hyponatremia, and hyperkalemia due to a renal tubular insensitivity to mineralocorticoids.假性肾上腺皮质功能减退症；由于肾小管对盐皮质激素不敏感导致肾钠丢失、低钠血症和高钾血症。
AMA J Dis Child. 1959 Jun;97(6):813-28.

A REVERSIBLE SALT-WASTING SYNDROME OF THE NEWBORN AND INFANT: POSSIBLE INFANTILE HYPOALDOSTERONISM.

作者信息

RUSSELL A, LEVIN B, SINCLAIR L, OBERHOLZER V G

出版信息

Arch Dis Child. 1963 Aug;38(200):313-25. doi: 10.1136/adc.38.200.313.

DOI:10.1136/adc.38.200.313
PMID:14058804
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2018943/
Abstract
摘要