胱氨酸尿症单一肾脏转运缺陷的反证 - Suppr

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EVIDENCE AGAINST A SINGLE RENAL TRANSPORT DEFECT IN CYSTINURIA.

作者信息

FOX M, THIER S, ROSENBERG L, KISER W, SEGAL S

出版信息

N Engl J Med. 1964 Mar 12;270:556-61. doi: 10.1056/NEJM196403122701105.

DOI:10.1056/NEJM196403122701105

PMID:14091528

Abstract

摘要

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引用本文的文献

TRANSPORT OF NEUTRAL AND DIBASIC AMINO ACIDS BY HUMAN LEUKOCYTES: ABSENCE OF DEFECT IN CYSTINURIA.

J Clin Invest. 1965 Aug;44(8):1382-93. doi: 10.1172/JCI105243.

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J Clin Invest. 1965 Mar;44(3):442-8. doi: 10.1172/JCI105157.

Cystine uptake by rat renal brush-border vesicles.

Biochem J. 1981 Feb 15;194(2):443-9. doi: 10.1042/bj1940443.

Reexamination of the interplay between dibasic amino acids and I-cystine/L-cysteine during tubular reabsorption.

Pflugers Arch. 1982 Nov 11;395(3):196-200. doi: 10.1007/BF00584809.

Dibasic amino acid transport: lessons from human disease.

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In vitro study of cysteine oxidase in rat brain.

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[An isolated defect of the tubular cystine reabsorption in a family with idiopathic hypoparathyroidism].

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The renal clearance of amino acids in cystinuria.

J Clin Invest. 1967 Jul;46(7):1162-71. doi: 10.1172/JCI105609.

Intestinal transport of cystine and cysteine in man: evidence for separate mechanisms.

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The intracellular cysteine-cystine ratio in kidney cortex.

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