Bordeianu C D
Service of Ophthalmologie, Ploieşti.
Oftalmologia. 1992 Oct-Dec;36(4):331-42.
On the basis of a pathogenic definition, our classification distinguished for each of the first intersections a main--mostly determinant mechanism and a secondary mechanism, mostly modulating the damages: hydrodynamic alteration and tissue resistance alteration--for the second intersection, outflow block and hypersecretion--for the third. Reflecting the state of present knowledge, from the corresponding forms of the disease, only outflow block glaucoma is completely analysed, the exogenous, trabecular, pretrabecular and mixed glaucomas being described. In order to permit the correct framing of all pretrabecular block glaucomas, the posterior push forms (retrovitreal or parietal) were introduced besides the anterior already described forms (pupillary or angular). As for low tissue resistance glaucomas, genetic, cardiovascular, metabolic, haematologic and ocular influences are mentioned. This classification avoids the theoretical inadvertencies of previous classifications offering the synthetic frame of a coherent system, open to any new developments, all inclusive, terminologically clear and with direct therapeutic references.
基于病因学定义，我们的分类为每个首要交叉点区分出一种主要（大多为决定性）机制和一种次要机制，后者大多调节损害：对于第一个交叉点，为流体动力学改变和组织阻力改变；对于第二个交叉点，为房水流出受阻和分泌过多；对于第三个交叉点，情况亦是如此。反映当前的知识状态，从疾病的相应类型来看，仅对房水流出受阻性青光眼进行了全面分析，对外源性、小梁性、小梁前性和混合性青光眼进行了描述。为了正确界定所有小梁前性阻滞性青光眼，除了已描述的前部类型（瞳孔性或房角性）外，还引入了后部推动形式（玻璃体后或壁部）。至于低组织阻力性青光眼，则提及了遗传、心血管、代谢、血液学和眼部影响。这种分类避免了以往分类在理论上的疏忽，提供了一个连贯系统的综合框架，对任何新进展开放，涵盖所有方面，术语清晰且具有直接的治疗参考价值。
