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热休克和紫外线B光对表皮角质形成细胞应激蛋白表达的不同影响。

Differential effects of heat shock and UVB light upon stress protein expression in epidermal keratinocytes.

作者信息

Maytin E V

机构信息

Department of Dermatology, Massachusetts General Hospital, Boston 02129.

出版信息

J Biol Chem. 1992 Nov 15;267(32):23189-96.

PMID:1429666
Abstract

Heat stress and ultraviolet light in the UVB range ("sunburn spectrum," 290-320 nm) were found to alter the synthesis of specific proteins in cultured keratinocytes derived from mouse skin. Using giant two-dimensional gels, approximately 2,000 cellular polypeptides labeled with [35S]methionine at 4-5 h after exposure to heat or to UVB were analyzed. Cells conditioned at sublethal temperatures (42 degrees C for 1 h, or 47 degrees C for 15 min) developed thermotolerance, while cells conditioned with UVB did not develop thermotolerance. Under these heat or UVB conditions, 19 stress proteins were observed. Proteins fell into three classes based upon their inducibility by heat or UVB, dose-response, and induction mechanism (transcriptional versus post-transcriptional) as defined by metabolic blockade with cordycepin (3'-deoxyadenosine). Class 1 proteins were inducible only by heat shock. They included three major heat-shock proteins (hsp 72, hsp 78, hsp 90) and a 42.5-kDa, pI 5.43 protein, and all were induced at the transcriptional level. Class 2 proteins were inducible by heat and by UVB. These included hsp 110 and eight additional polypeptides. All but one were affected by heat at the post-transcriptional level and were induced by UVB at both low (20 mJ/cm2) and high (80 mJ/cm2) doses. Class 3 proteins were inducible only at high UVB doses (survival < 10%). Class 1 and Class 2 proteins could be functionally involved in thermotolerance, while Class 3 proteins are more likely related to damage or cell death.

摘要

研究发现,热应激以及紫外线B波段(“晒伤光谱”,290 - 320纳米)的紫外线会改变从小鼠皮肤分离培养的角质形成细胞中特定蛋白质的合成。利用大型二维凝胶,对在暴露于热或紫外线B后4 - 5小时用[35S]甲硫氨酸标记的约2000种细胞多肽进行了分析。在亚致死温度(42℃处理1小时,或47℃处理15分钟)下培养的细胞产生了耐热性,而用紫外线B处理的细胞未产生耐热性。在这些热或紫外线B条件下,观察到了19种应激蛋白。根据热或紫外线B的诱导性、剂量反应以及诱导机制(通过用虫草素(3'-脱氧腺苷)进行代谢阻断来定义转录与转录后),蛋白质分为三类。第1类蛋白质仅可由热休克诱导。它们包括三种主要的热休克蛋白(热休克蛋白72、热休克蛋白78、热休克蛋白90)以及一种42.5 kDa、等电点为5.43的蛋白质,并且所有这些蛋白质均在转录水平上被诱导。第2类蛋白质可由热和紫外线B诱导。这些包括热休克蛋白110和另外8种多肽。除一种外,所有这些蛋白质在转录后水平受热影响,并且在低剂量(20 mJ/cm2)和高剂量(80 mJ/cm2)紫外线B照射下均被诱导。第3类蛋白质仅在高剂量紫外线B照射下(存活率<10%)被诱导。第1类和第2类蛋白质可能在功能上与耐热性有关,而第3类蛋白质更可能与损伤或细胞死亡有关。

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