Leduc D, Gris P, Lheureux P, Gevenois P A, De Vuyst P, Yernault J C
Department of Chest Medicine, Erasme Hospital, Free University of Brussels, Belgium.
Thorax. 1992 Sep;47(9):755-7. doi: 10.1136/thx.47.9.755.
A lifelong non-smoker who was the victim of a massive accidental exposure to anhydrous ammonia gas was followed up for 10 years. In the acute phase the patient presented with severe tracheobronchitis and respiratory failure, caused by very severe burns of the respiratory mucosa. After some improvement he was left with severe and fixed airways obstruction. Isotope studies of mucociliary clearance, computed tomography, and bronchography showed mild bronchiectasis. It is concluded that acute exposure to high concentrations of ammonia may lead to acute respiratory injury but also to long term impairment of respiratory function.
一名终生不吸烟者因意外大量接触无水氨气而成为受害者，对其进行了10年的随访。急性期患者出现严重的气管支气管炎和呼吸衰竭，这是由呼吸道黏膜的严重烧伤引起的。经过一段时间的改善后，他仍留有严重且固定的气道阻塞。黏液纤毛清除的同位素研究、计算机断层扫描和支气管造影显示有轻度支气管扩张。得出的结论是，急性接触高浓度氨气可能导致急性呼吸损伤，也会导致呼吸功能的长期损害。
