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实验性半乳糖性白内障中的酶因素

Enzymatic factors in experimental galactose cataract.

作者信息

LERMAN S

出版信息

Science. 1959 Nov 27;130(3387):1473-4. doi: 10.1126/science.130.3387.1473-a.

DOI:10.1126/science.130.3387.1473-a
PMID:14415929
Abstract

In the rat lens, glucose-6-phosphate dehydrogenase activity is specifically inhibited by galactose-l-phosphate, both in vitro and in vivo. In galactose-fed animals this inhibition occurs prior to the apparent inhibition of soluble protein synthesis in the ocular lens.

摘要

在大鼠晶状体中,6-磷酸葡萄糖脱氢酶活性在体外和体内均受到1-磷酸半乳糖的特异性抑制。在喂食半乳糖的动物中,这种抑制发生在眼晶状体中可溶性蛋白质合成明显受到抑制之前。

相似文献

1
Enzymatic factors in experimental galactose cataract.实验性半乳糖性白内障中的酶因素
Science. 1959 Nov 27;130(3387):1473-4. doi: 10.1126/science.130.3387.1473-a.
2
[ACTIVITY OF SOME ENZYMES ON THE CRYSTALLINE LENS OF NORMAL RATS OF DIFFERENT AGES. GLUCOSE-6-PHOSPHATE DEHYDROGENASE AND ALDOLASE].
An Fac Med Univ Repub Montev Urug. 1963;48:335-8.
3
Effect of galactose-1-phosphate on glucose oxidation by normal and galactosemic leukocytes.
Science. 1960 Oct 14;132(3433):1015-6. doi: 10.1126/science.132.3433.1015.
4
[Structure and formation of fibers in the normal crystalline lens. Their modification in galactose cataract. Electron microscope study in the rat].[正常晶状体中纤维的结构与形成。半乳糖性白内障中纤维的改变。大鼠的电子显微镜研究]
Bull Soc Ophtalmol Fr. 1961 May-Jun;5:340-3.
5
Aldose reductase, NADPH and NADP+ in normal, galactose-fed and diabetic rat lens.正常、喂食半乳糖及糖尿病大鼠晶状体中的醛糖还原酶、还原型辅酶II和辅酶II
Biochim Biophys Acta. 1985 Sep 6;841(3):247-53. doi: 10.1016/0304-4165(85)90065-0.
6
[Variation of some enzymatic activities of the rat crystalline lens in experimental galactose cataract (lactate dehydrogenase, leucine aminopeptidase, phosphoglucose isomerase].实验性半乳糖性白内障大鼠晶状体某些酶活性的变化(乳酸脱氢酶、亮氨酸氨肽酶、磷酸葡萄糖异构酶)
C R Seances Soc Biol Fil. 1968;162(2):435-7.
7
Hypophysectomy effect on lens epithelium mitosis and galactose cataract development in rats.垂体切除对大鼠晶状体上皮细胞有丝分裂及半乳糖性白内障形成的影响。
Arch Ophthalmol. 1962 Apr;67:476-82. doi: 10.1001/archopht.1962.00960020476016.
8
Antioxidant potential of fungal metabolite nigerloxin during eye lens abnormalities in galactose-fed rats.在半乳糖喂养的大鼠的眼睛晶状体异常中真菌代谢产物虎奶菇素的抗氧化潜力。
Curr Eye Res. 2013 Oct;38(10):1064-71. doi: 10.3109/02713683.2013.802810. Epub 2013 Jun 14.
9
Enzymatic studies on the galactose exposed lens.对半乳糖暴露晶状体的酶学研究。
Indian J Ophthalmol. 1980 Apr;28(1):5-7.
10
High galactose levels in vitro and in vivo impair ascorbate regeneration and increase ascorbate-mediated glycation in cultured rat lens.体外和体内的高半乳糖水平会损害培养的大鼠晶状体中的抗坏血酸再生,并增加抗坏血酸介导的糖基化反应。
Exp Eye Res. 1996 Nov;63(5):535-45. doi: 10.1006/exer.1996.0144.

引用本文的文献

1
COMPLEMENTATION, RECOMBINATION, AND SUPPRESSION IN GALACTOSE NEGATIVE MUTANTS OF E. COLI.大肠杆菌半乳糖阴性突变体中的互补、重组和抑制作用
Proc Natl Acad Sci U S A. 1963 Aug;50(2):286-93. doi: 10.1073/pnas.50.2.286.
2
Oxidative hemolysis and precipitation of hemoglobin. I. Heinz body anemias as an acceleration of red cell aging.氧化溶血与血红蛋白沉淀。I. 海因茨小体贫血作为红细胞衰老的加速过程。
J Clin Invest. 1960 Dec;39(12):1818-36. doi: 10.1172/JCI104206.
3
The metabolism of xylose by the ocular lens of the rat.大鼠晶状体对木糖的代谢
Biochem J. 1961 May;79(2):224-9. doi: 10.1042/bj0790224.
4
[Glutathione peroxidase: enzymology and biological aspects].[谷胱甘肽过氧化物酶:酶学与生物学方面]
Klin Wochenschr. 1971 Jun 15;49(12):669-83. doi: 10.1007/BF01487101.
5
Further data on the effect of galactose on bone tissue.关于半乳糖对骨组织影响的更多数据。
Calcif Tissue Res. 1974;16(2):153-5. doi: 10.1007/BF02008221.
6
The effect of galactose on bone metabolism.半乳糖对骨代谢的影响。
Calcif Tissue Res. 1974 Mar 29;14(3):169-75. doi: 10.1007/BF02060293.