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Acute effects of methoxypyridoxine on hippocampal end-blade neurons: an experimental study of "special pathoclisis" in the cerebral cortex.

作者信息

PURPURA D P, GONZALEZ-MONTEAGUDO O

出版信息

J Neuropathol Exp Neurol. 1960 Jul;19:421-32. doi: 10.1097/00005072-196007000-00009.

DOI:10.1097/00005072-196007000-00009
PMID:14435353
Abstract
摘要

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1
Acute effects of methoxypyridoxine on hippocampal end-blade neurons: an experimental study of "special pathoclisis" in the cerebral cortex.
J Neuropathol Exp Neurol. 1960 Jul;19:421-32. doi: 10.1097/00005072-196007000-00009.
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Modification of cortical, reticular and hippocampal unit activity by nicotine in the rabbit.
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[Action of nicotine on the bioelectric activity of the cortex of the thalamus and of the hippocampus in rabbits. Its "arousal" action and primary convulsive action on the hippocampo-thalamic structures].
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Low distribution of synaptic vesicle protein 2A and synaptotagimin-1 in the cerebral cortex and hippocampus of spontaneously epileptic rats exhibiting both tonic convulsion and absence seizure.在表现出强直痉挛和失神发作的自发性癫痫大鼠的大脑皮层和海马体中，突触小泡蛋白 2A 和突触结合蛋白-1 的分布较低。
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Some effects of antagonists and analogs of vitamin B6 in yeast.维生素B6拮抗剂及类似物在酵母中的某些作用。
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9
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Kainic acid seizures cause enhanced expression of cholecystokinin-octapeptide in the cortex and hippocampus of the rat.海人酸诱发的癫痫发作会导致大鼠大脑皮层和海马体中胆囊收缩素八肽的表达增强。
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Classic hippocampal sclerosis and hippocampal-onset epilepsy produced by a single "cryptic" episode of focal hippocampal excitation in awake rats.清醒大鼠单一“隐匿性”海马局部兴奋发作致经典海马硬化和海马起始性癫痫
J Comp Neurol. 2010 Aug 15;518(16):3381-407. doi: 10.1002/cne.22406.
2
[Problems in pathologic anatomy in temporal lobe epilepsy].[颞叶癫痫的病理解剖学问题]
Dtsch Z Nervenheilkd. 1962;183:245-57.
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[CLINICAL AND PATHOMORPHOLOGICAL FINDINGS IN CHRONIC ADDICTION TO ANTIASTHMATIC DRUGS].[慢性抗哮喘药物成瘾的临床及病理形态学表现]
Arch Psychiatr Nervenkr. 1964 Jun 1;205:306-20. doi: 10.1007/BF00395718.
4
Cycloheximide prevents kainate-induced neuronal death and c-fos expression in adult rat brain.放线菌酮可预防成年大鼠脑中红藻氨酸诱导的神经元死亡和c-fos表达。
J Mol Neurosci. 1993 Fall;4(3):149-59. doi: 10.1007/BF02782498.
5
Pathogenesis of brain lesions caused by experimental epilepsy. Light- and electron-microscopic changes in the rat cerebral cortex following bicuculline-induced status epilepticus.实验性癫痫所致脑损伤的发病机制。荷包牡丹碱诱发癫痫持续状态后大鼠大脑皮质的光镜和电镜变化。
Acta Neuropathol. 1981;54(3):219-31. doi: 10.1007/BF00687745.
6
Bicuculline-induced epileptic brain injury. Transient and persistent cell changes in rat cerebral cortex in the early recovery period.荷包牡丹碱诱导的癫痫性脑损伤。大鼠大脑皮层在早期恢复阶段的短暂和持续性细胞变化。
Acta Neuropathol. 1983;62(1-2):87-95. doi: 10.1007/BF00684924.
7
Pathogenesis of brain lesions caused by experimental epilepsy. Light- and electron-microscopic changes in the rat hippocampus following bicuculline-induced status epilepticus.实验性癫痫所致脑损伤的发病机制。荷包牡丹碱诱导癫痫持续状态后大鼠海马的光镜和电镜变化。
Acta Neuropathol. 1983;59(1):11-24. doi: 10.1007/BF00690312.
8
The histochemical architecture of the Ammon's horn as related to its selective vulnerability.
Acta Neuropathol. 1966 Jan 14;6(1):1-13. doi: 10.1007/BF00691077.
9
Selective vulnerability of brain: new insights from the excitatory synapse.大脑的选择性易损性：来自兴奋性突触的新见解。
Metab Brain Dis. 1986 Dec;1(4):231-40. doi: 10.1007/BF00999353.
10
Golgi-like demonstration of "dark" neurons with an argyrophil III method for experimental neuropathology.采用嗜银III法对实验性神经病理学中“深色”神经元进行高尔基样显示。
Acta Neuropathol. 1990;79(6):620-8. doi: 10.1007/BF00294239.