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[切尔诺贝利核电站事故后遗症清理参与者中伴有神经循环性肌张力障碍综合征的调节碳水化合物代谢的激素功能]

[The hormonal functions regulating carbohydrate metabolism in participants in the cleanup of the sequelae of the accident at the Chernobyl Atomic Electric Power Station with a neurocirculatory dystonia syndrome].

作者信息

Kovalenko A N, Sushko V A, Fedirko M I

出版信息

Lik Sprava. 1992 Jun(6):52-5.

PMID:1455829
Abstract

Radioimmunoassay revealed in participants of Chernobyl disaster sequelae preclinical changes of some hormonal functions: moderate increase of the basal concentration of blood insulin, somatotropin, C-peptide against the background of persistent hypercortisolemia. Increase of the level of "hyperglycemic" hormones (cortisol, somatotropin, C-peptide) is explained by adaptative and compensatory reactions of the body in response to lesioning effects of ionizing radiation and other negative factors of the Chernobyl accident. Relative hyperinsulinemia is, apparently, compensating the total hyperglycemic effect of these hormones leading to stabilization of the blood sugar level.

摘要

放射免疫分析显示，切尔诺贝利灾难后遗症患者存在一些激素功能的临床前变化：在持续性高皮质醇血症的背景下，血液胰岛素、生长激素、C肽的基础浓度适度升高。“升糖”激素（皮质醇、生长激素、C肽）水平的升高是身体对电离辐射损伤效应及切尔诺贝利事故其他负面因素的适应性和代偿性反应所致。相对高胰岛素血症显然是在代偿这些激素的总体升糖作用，从而使血糖水平趋于稳定。

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